Literature DB >> 17054696

SCN1A mutation mosaicism in a family with severe myoclonic epilepsy in infancy.

Masafumi Morimoto1, Emi Mazaki, Akira Nishimura, Tomohiro Chiyonobu, Yasuko Sawai, Aki Murakami, Keiko Nakamura, Ikuyo Inoue, Ikuo Ogiwara, Tohru Sugimoto, Kazuhiro Yamakawa.   

Abstract

PURPOSE: To investigate the genetic background of familial severe myoclonic epilepsy in infancy (SMEI) cases.
METHODS: We performed mutation analyses of the sodium-channel gene SCN1A in two Japanese brothers with clinical features of SMEI and their parents, who had no history of febrile and epileptic seizures.
RESULTS: Each patient showed nucleotide changes (c.[730G>T; 735G>T; 736A>T]) in the coding exon 6 of SCN1A that led to a truncation of the channel protein. Their father showed no mutations, but their mother showed the same mutation in a subpopulation of lymphocytes.
CONCLUSIONS: The maternal mosaicism explains the identical SCN1A mutations in the two brothers. This highlights the importance of investigating parental mosaicism even in sporadic SMEI cases.

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Year:  2006        PMID: 17054696     DOI: 10.1111/j.1528-1167.2006.00645.x

Source DB:  PubMed          Journal:  Epilepsia        ISSN: 0013-9580            Impact factor:   5.864


  15 in total

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