Literature DB >> 17031491

P38 MAPK protects against TNF-alpha-provoked apoptosis in LNCaP prostatic cancer cells.

M Ricote1, I García-Tuñón, B Fraile, C Fernández, P Aller, R Paniagua, M Royuela.   

Abstract

PURPOSE: One of the most relevant aspects in cell death regulation is the signalling of apoptosis by the serine/threonine kinases MAPKs. The aim of this study was to investigate the effects of TNF-alpha stimulation on MAPK activation, and the pro- or anti-apoptotic role of these kinases in LNCaP and PC3 cells.
MATERIAL AND METHODS: Treatments were carried out using several TNF-alpha concentrations, as well as specific pharmacological inhibitors of MAPKs. Apoptosis rates were evaluated by DAPI staining and flow cytometry. MAPK phosphorylation/activation was measured by Western blot.
RESULTS: TNF-alpha induced apoptosis in a dose-dependent manner in LNCaP but not in PC3 cells. The MAPK inhibitors revealed that the apoptotic rate in LNCaP cells increased significantly following p38 inhibition. The kinase inhibitors failed to cause changes in apoptosis in PC3 cells.
CONCLUSIONS: The potentiation of apoptosis by p38 inhibition points to this kinase as a possible target for the treatment of androgen-dependent prostatic cancer.

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Year:  2006        PMID: 17031491     DOI: 10.1007/s10495-006-0086-9

Source DB:  PubMed          Journal:  Apoptosis        ISSN: 1360-8185            Impact factor:   4.677


  12 in total

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6.  MAP Kinases and Prostate Cancer.

Authors:  Gonzalo Rodríguez-Berriguete; Benito Fraile; Pilar Martínez-Onsurbe; Gabriel Olmedilla; Ricardo Paniagua; Mar Royuela
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9.  Reduced tumor growth in vivo and increased c-Abl activity in PC3 prostate cancer cells overexpressing the Shb adapter protein.

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