Literature DB >> 17005169

Immunoassay of serine-phosphorylated isoform of insulin-like growth factor (IGF) binding protein (IGFBP)-1.

Javad Khosravi1, Radha G Krishna, Umesh Bodani, Anastasia Diamandi, Najmuddin Khaja, Bhanu Kalra, Ajay Kumar.   

Abstract

OBJECTIVES: Development of an ELISA for phosphorylated isoform of IGFBP-1. Serine phosphorylation is an important regulator of IGFBP-1 bioactivity, but specific immunoassays for its measurement are currently lacking. DESIGN AND METHODS: Assay design was based on a novel approach of first capturing the phosphorylated and non-phosphorylated IGFBP-1 by an anti-IGFBP-1 antibody and then selectively detecting the phosphorylated form by an anti-phosphoserine antibody. Method development involved pair-wise evaluation of the candidate antibodies and determinations of analytical performance and specificity. Specificity was monitored by reactivity with dephosphorylated IGFBP-1, with antibodies against other phosphorylated residues that are not expressed, and by comparative analysis of sample containing different IGFBP-1 phosphorylation profile.
RESULTS: Analytical evaluation demonstrated acceptable performance; detection limit 0.3 microg/L, dynamic range 1.56-100 microg/L; intra- and inter-assay CVs 2.1-8.6%; mean recovery (+/-SD) 97.8+/-9.2%, and mean recovery of sample dilution 93.4+/-6.0%. The phosphorylated and total IGFBP-1 medians in non-pregnant adult serum, which mostly contain the highly phosphorylated isoform, were 11.9 and 18.6 microg/L, respectively, and the sample values were tightly correlated (r=0.99). As expected, the corresponding medians in 1st trimester (17.4 and 63.0 microg/L) and 2nd trimester (30.9 and 75.8) samples with altered IGFBP-1 phosphorylation were significantly different (p<0.001). Similarly, a fraction (1.29%) of total IGFBP-1 (13.3 mg/L) in amniotic fluids was found to be phosphorylated (0.172 mg/L). There was no reactivity with dephosphorylated IGFBP-1.
CONCLUSIONS: The present ELISA is highly specific for the phosphorylated isoform of IGFBP-1 and its availability should help expedite further investigations of IGFBP-1 phosphorylation.

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Year:  2006        PMID: 17005169     DOI: 10.1016/j.clinbiochem.2006.07.004

Source DB:  PubMed          Journal:  Clin Biochem        ISSN: 0009-9120            Impact factor:   3.281


  5 in total

1.  The role and regulation of IGFBP-1 phosphorylation in fetal growth restriction.

Authors:  Madhulika B Gupta
Journal:  J Cell Commun Signal       Date:  2015-02-15       Impact factor: 5.782

2.  Hypoxia and leucine deprivation induce human insulin-like growth factor binding protein-1 hyperphosphorylation and increase its biological activity.

Authors:  Maxim D Seferovic; Rashad Ali; Hiroyasu Kamei; Suya Liu; Javad M Khosravi; Steven Nazarian; Victor K M Han; Cunming Duan; Madhulika B Gupta
Journal:  Endocrinology       Date:  2008-09-04       Impact factor: 4.736

3.  Functional and complementary phosphorylation state attributes of human insulin-like growth factor-binding protein-1 (IGFBP-1) isoforms resolved by free flow electrophoresis.

Authors:  Mikkel Nissum; Majida Abu Shehab; Ute Sukop; Javad M Khosravi; Robert Wildgruber; Christoph Eckerskorn; Victor K M Han; Madhulika B Gupta
Journal:  Mol Cell Proteomics       Date:  2009-02-03       Impact factor: 5.911

4.  Insulin-like growth factor I (IGF-I) as a sensitive biomarker of catabolism in patients with gastrointestinal diseases.

Authors:  Olgica Nedić; Vesna Malenković; Judith Anna Nikolić; Ivona Baricević
Journal:  J Clin Lab Anal       Date:  2007       Impact factor: 2.352

Review 5.  Amniotic fluid embolism pathophysiology suggests the new diagnostic armamentarium: β-tryptase and complement fractions C3-C4 are the indispensable working tools.

Authors:  Francesco Paolo Busardò; Paola Frati; Simona Zaami; Vittorio Fineschi
Journal:  Int J Mol Sci       Date:  2015-03-23       Impact factor: 5.923

  5 in total

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