| Literature DB >> 1694829 |
Abstract
Tumor necrosis factor alpha (TNF alpha) has been implicated as a major mediator of lipopolysaccharide (LPS)-induced phenomena. Administration to mice of a polyclonal, monospecific antibody prepared against recombinant murine TNF alpha abolished detection of LPS-induced TNF alpha activity and significantly reduced levels of LPS-induced colony-stimulating factor but failed to reduce the production of LPS-induced interferon, corticosterone, or LPS-induced hypoglycemia.Entities:
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Year: 1990 PMID: 1694829 PMCID: PMC258827 DOI: 10.1128/iai.58.7.2397-2400.1990
Source DB: PubMed Journal: Infect Immun ISSN: 0019-9567 Impact factor: 3.441