| Literature DB >> 1694126 |
D Wolf1, D Quon, Y Wang, B Cordell.
Abstract
The mechanism of amyloid formation in Alzheimer's disease is unknown but appears to involve proteolytic processing of the amyloidogenic peptide from a larger precursor. When the C-terminus containing the amyloid-forming and cytoplasmic domains of the precursor was recombinantly expressed in cultured mammalian cells, a 16 kd protein accumulated which had a tendency to aggregate and form deposits. These deposits had physical characteristics resembling those of preamyloid. Recombinant expression of the full-length precursor was found to produce a similar, cell-associated 16 kd C-terminal fragment as well as a 12 kd fragment, neither of which formed detectable aggregates suggesting efficient catabolism of these fragments. Identification of these two naturally occurring metabolic products of the beta-amyloid precursor provides a system permitting the characterization of the proteolytic processing events of the amyloid precursor protein.Entities:
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Year: 1990 PMID: 1694126 PMCID: PMC551925 DOI: 10.1002/j.1460-2075.1990.tb07375.x
Source DB: PubMed Journal: EMBO J ISSN: 0261-4189 Impact factor: 11.598