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14 in total

1. Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria.

Authors: A J Kowaltowski; S Seetharaman; P Paucek; K D Garlid
Journal: Am J Physiol Heart Circ Physiol Date: 2001-02 Impact factor: 4.733

2. Roles of mitochondrial ATP-sensitive K channels and PKC in anti-infarct tolerance afforded by adenosine A1 receptor activation.

Authors: T Miura; Y Liu; H Kita; T Ogawa; K Shimamoto
Journal: J Am Coll Cardiol Date: 2000-01 Impact factor: 24.094

3. Inhibition of mitochondrial metabolism by the diabetogenic thiadiazine diazoxide. II. Interaction with energy conservation and ion transport.

Authors: R Portenhauser; G Schäfer; R Trolp
Journal: Biochem Pharmacol Date: 1971-10 Impact factor: 5.858

4. Diazoxide and pinacidil uncouple pyruvate-malate-induced mitochondrial respiration.

Authors: Dalia M Kopustinskiene; Jovita Jovaisiene; Julius Liobikas; Adolfas Toleikis
Journal: J Bioenerg Biomembr Date: 2002-02 Impact factor: 2.945

5. Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning.

Authors: Paavo Korge; Henry M Honda; James N Weiss
Journal: Proc Natl Acad Sci U S A Date: 2002-02-26 Impact factor: 11.205

6. Mitochondrial Ca2+-activated K+ channels in cardiac myocytes: a mechanism of the cardioprotective effect and modulation by protein kinase A.

Authors: Toshiaki Sato; Tomoaki Saito; Noriko Saegusa; Haruaki Nakaya
Journal: Circulation Date: 2004-12-27 Impact factor: 29.690

7. Opening of mitochondrial KATP channels enhances cardioprotection through the modulation of mitochondrial matrix volume, calcium accumulation, and respiration.

Authors: Anthony J Rousou; Maria Ericsson; Micheline Federman; Sidney Levitsky; James D McCully
Journal: Am J Physiol Heart Circ Physiol Date: 2004-07-08 Impact factor: 4.733

3. Closure of mitochondrial potassium channels favors opening of the Tl(+)-induced permeability transition pore in Ca(2+)-loaded rat liver mitochondria.

Authors: Sergey M Korotkov; Irina V Brailovskaya; Anton R Shumakov; Larisa V Emelyanova
Journal: J Bioenerg Biomembr Date: 2015-04-14 Impact factor: 2.945

3 in total

Study of the mechanism of action of diazoxide on rat heart mitochondria under calcium loading.

1. Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria.

2. Roles of mitochondrial ATP-sensitive K channels and PKC in anti-infarct tolerance afforded by adenosine A1 receptor activation.

3. Inhibition of mitochondrial metabolism by the diabetogenic thiadiazine diazoxide. II. Interaction with energy conservation and ion transport.

4. Diazoxide and pinacidil uncouple pyruvate-malate-induced mitochondrial respiration.

5. Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning.

6. Mitochondrial Ca2+-activated K+ channels in cardiac myocytes: a mechanism of the cardioprotective effect and modulation by protein kinase A.

7. Opening of mitochondrial KATP channels enhances cardioprotection through the modulation of mitochondrial matrix volume, calcium accumulation, and respiration.

8. Potassium channel openers are uncoupling protonophores: implication in cardioprotection.

9. Diazoxide affects the IF1 inhibitor protein binding to F1 sector of beef heart F0F1ATPsynthase.

10. K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heart.

1. Involvement of SH-groups during interaction of diazoxide with the inner membrane of rat heart mitochondria.

2. Effect of diazoxide and Ca2+ on rat heart mitochondria loaded with Na+.

3. Closure of mitochondrial potassium channels favors opening of the Tl(+)-induced permeability transition pore in Ca(2+)-loaded rat liver mitochondria.