Literature DB >> 16801331

Association of mannose-binding lectin gene polymorphisms with antiphospholipid syndrome, cardiovascular disease and chronic damage in patients with systemic lupus erythematosus.

J Font1, M Ramos-Casals, P Brito-Zerón, N Nardi, A Ibañez, B Suarez, S Jiménez, D Tàssies, A García-Criado, E Ros, J Sentís, J-C Reverter, F Lozano.   

Abstract

OBJECTIVE: To investigate the association of mannose-binding lectin (MBL)-deficient genotypes with cardiovascular disease in a large series of patients with systemic lupus erythematosus (SLE).
METHODS: A total of 114 patients diagnosed with SLE were included in the study. MBL polymorphisms were investigated by sequencing-based DNA typing of the promoter and exon 1 of the MBL2 gene. The genotypes 0/0, 0/XA and XA/XA were considered as MBL-low genotypes.
RESULTS: A higher prevalence of cardiovascular disease was observed in patients carrying MBL-low genotypes compared with those carrying MBL-high genotypes [30 vs 9%, P = 0.012, odds ratio (OR) 4.54, 95% confidence interval (CI) 1.20-16.46]. Patients with MBL-low genotypes also presented higher mean values for total cholesterol (228.6 vs 202.3 mg/dl, P = 0.017) and low-density lipoprotein (LDL) cholesterol (139.9 vs 121.9 mg/dl, P = 0.045), a higher frequency of chronic renal failure (30 vs 4%, P = 0.001), vasculitis (30 vs 11%, P = 0.043), heart valve lesions (71 vs 32%, P = 0.026), cardiac valve dysfunction (57 vs 7%, P = 0.0004) and associated APS (39 vs 12%, P = 0.005), a higher mean Systemic Lupus International Collaborating Clinics score (2.09 vs 1.26, P = 0.029) and a lower prevalence of low C4 levels (43 vs 71%, P = 0.015). Multivariate analysis of genetic, clinical and immunological variables showed that only antiphospholipid syndrome (APS) was independently associated with cardiovascular events (P = 0.001).
CONCLUSION: Although the prevalence of cardiovascular disease in our SLE patients carrying MBL-deficient genotypes was 3.3 times higher than in patients with non-deficient genotypes, only APS was independently associated with cardiovascular events. This suggests that the higher frequency of thrombotic events in SLE patients carrying MBL-deficient genotypes might be related to coexisting APS.

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Year:  2006        PMID: 16801331     DOI: 10.1093/rheumatology/kel199

Source DB:  PubMed          Journal:  Rheumatology (Oxford)        ISSN: 1462-0324            Impact factor:   7.580


  11 in total

1.  Mannose-binding lectin (MBL) codon 54 (rs1800450) polymorphism predisposes towards medium vessel vasculitis in patients with systemic lupus erythematosus.

Authors:  Vir Singh Negi; Panneer Devaraju; Durga Prasanna Misra; Vikramraj K Jain; Jignesh Babulal Usdadiya; Paul T Antony; Reena Gulati
Journal:  Clin Rheumatol       Date:  2017-01-17       Impact factor: 2.980

2.  Patterns and levels of platelet glycosylation in patients with coronary heart disease and type 2 diabetes mellitus.

Authors:  Liping Li; Chenxue Qu; Xuelian Wu; Juhua Dai; Yao Lu; Yan Gong; Ran You; Yaqi Liu
Journal:  J Thromb Thrombolysis       Date:  2018-01       Impact factor: 2.300

3.  Mannose-binding lectin genotype and serum levels in patients with chronic and allergic pulmonary aspergillosis.

Authors:  E Harrison; A Singh; J Morris; N L Smith; M G Fraczek; C B Moore; D W Denning
Journal:  Int J Immunogenet       Date:  2012-01-09       Impact factor: 1.466

Review 4.  Global trends, potential mechanisms and early detection of organ damage in SLE.

Authors:  Anselm Mak; David A Isenberg; Chak-Sing Lau
Journal:  Nat Rev Rheumatol       Date:  2012-12-11       Impact factor: 20.543

Review 5.  The role of mannose-binding lectin in systemic lupus erythematosus.

Authors:  Odirlei André Monticielo; Tamara Mucenic; Ricardo Machado Xavier; João Carlos Tavares Brenol; José Artur Bogo Chies
Journal:  Clin Rheumatol       Date:  2008-01-24       Impact factor: 2.980

6.  Association of mannose-binding lectin gene polymorphism but not of mannose-binding serine protease 2 with chronic severe aortic regurgitation of rheumatic etiology.

Authors:  Rajendranath Ramasawmy; Guilherme S Spina; Kellen C Fae; Alexandre C Pereira; Renato Nisihara; Iara Jose Messias Reason; Max Grinberg; Flavio Tarasoutchi; Jorge Kalil; Luiza Guilherme
Journal:  Clin Vaccine Immunol       Date:  2008-04-09

7.  Fluorochrome-linked immunoassay for functional analysis of the mannose binding lectin complement pathway to the level of C3 cleavage.

Authors:  Mary C Walsh; Lisa A Shaffer; Benjamin J Guikema; Simon C Body; Stanton K Shernan; Amanda A Fox; Charles D Collard; Michael Fung; Ronald P Taylor; Gregory L Stahl
Journal:  J Immunol Methods       Date:  2007-05-11       Impact factor: 2.303

8.  Association between low levels of Mannan-binding lectin and markers of autoimmune thyroid disease in pregnancy.

Authors:  Eliska Potlukova; Tomas Freiberger; Zdenka Limanova; Jan Jiskra; Zdenek Telicka; Jana Bartakova; Drahomira Springer; Hana Vitkova; Marten Trendelenburg
Journal:  PLoS One       Date:  2013-12-10       Impact factor: 3.240

Review 9.  Protective molecules and their cognate antibodies: new players in autoimmunity.

Authors:  Margherita Zen; Nicola Bassi; Carla Campana; Silvano Bettio; Elena Tarricone; Linda Nalotto; Anna Ghirardello; Andrea Doria
Journal:  Auto Immun Highlights       Date:  2010-11-04

10.  Opposite Profiles of Complement in Antiphospholipid Syndrome (APS) and Systemic Lupus Erythematosus (SLE) Among Patients With Antiphospholipid Antibodies (aPL).

Authors:  Stephanie L Savelli; Robert A S Roubey; Kathryn J Kitzmiller; Danlei Zhou; Haikady N Nagaraja; Evan Mulvihill; Fatima Barbar-Smiley; Stacy P Ardoin; Yee Ling Wu; Chack-Yung Yu
Journal:  Front Immunol       Date:  2019-05-07       Impact factor: 7.561

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