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18 in total

1. Phosphorylation of threonine 156 of the mu2 subunit of the AP2 complex is essential for endocytosis in vitro and in vivo.

Authors: O Olusanya; P D Andrews; J R Swedlow; E Smythe
Journal: Curr Biol Date: 2001-06-05 Impact factor: 10.834

2. Molecular architecture and functional model of the endocytic AP2 complex.

Authors: Brett M Collins; Airlie J McCoy; Helen M Kent; Philip R Evans; David J Owen
Journal: Cell Date: 2002-05-17 Impact factor: 41.582

3. The 14-3-3 protein translates the NA+,K+-ATPase {alpha}1-subunit phosphorylation signal into binding and activation of phosphoinositide 3-kinase during endocytosis.

Authors: Riad Efendiev; Zongpei Chen; Rafael T Krmar; Sabine Uhles; Adrian I Katz; Carlos H Pedemonte; Alejandro M Bertorello
Journal: J Biol Chem Date: 2005-02-18 Impact factor: 5.157

4. The interaction of epsin and Eps15 with the clathrin adaptor AP-2 is inhibited by mitotic phosphorylation and enhanced by stimulation-dependent dephosphorylation in nerve terminals.

Authors: H Chen; V I Slepnev; P P Di Fiore; P De Camilli
Journal: J Biol Chem Date: 1999-02-05 Impact factor: 5.157

5. Hypoxia-induced endocytosis of Na,K-ATPase in alveolar epithelial cells is mediated by mitochondrial reactive oxygen species and PKC-zeta.

Authors: Laura A Dada; Navdeep S Chandel; Karen M Ridge; Carlos Pedemonte; Alejandro M Bertorello; Jacob I Sznajder
Journal: J Clin Invest Date: 2003-04 Impact factor: 14.808

6. CK2 and GAK/auxilin2 are major protein kinases in clathrin-coated vesicles.

Authors: Viktor I Korolchuk; George Banting
Journal: Traffic Date: 2002-06 Impact factor: 6.215

7. Phosphorylation of adaptor protein-2 mu2 is essential for Na+,K+-ATPase endocytosis in response to either G protein-coupled receptor or reactive oxygen species.

Authors: Zongpei Chen; Rafael T Krmar; Laura Dada; Riad Efendiev; Ingo B Leibiger; Carlos H Pedemonte; Adrian I Katz; Jacob I Sznajder; Alejandro M Bertorello
Journal: Am J Respir Cell Mol Biol Date: 2006-02-23 Impact factor: 6.914

Review 8. Physiological role of the alpha1- and alpha2-isoforms of the Na+-K+-ATPase and biological significance of their cardiac glycoside binding site.

Authors: Iva Dostanic-Larson; John N Lorenz; James W Van Huysse; Jon C Neumann; Amy E Moseley; Jerry B Lingrel
Journal: Am J Physiol Regul Integr Comp Physiol Date: 2006-03 Impact factor: 3.619

2. Chronic insulin treatment phosphorylates the renal Na-K-ATPase α1-subunit at serine 16/23 and reduces its activity involving PI3-kinase-dependent PKC activation.

Authors: Anees Ahmad Banday
Journal: Am J Physiol Renal Physiol Date: 2016-09-07

2 in total

Unlocking the mysteries of Na+-K+-ATPase endocytosis: phosphorylation is the key.