Literature DB >> 1677605

N-terminal sequence of prion protein is also integrated into kuru plaques in patients with Gerstmann-Sträussler syndrome.

T Kitamoto1, T Muramoto, C Hilbich, K Beyreuther, J Tateishi.   

Abstract

Kuru plaques are one of the pathological hallmarks in Gerstmann-Sträussler syndrome, and are composed of prion protein (PrP). To elucidate whether N-terminal sequence of PrP is related to amyloid formation in vivo, we prepared antibody against synthetic N-terminal peptide (anti-PrP-N). Anti-PrP-N immunolabeled kuru plaques positively. Positive reactions were observed in the periphery of large kuru plaque cores, but not in the center. It is therefore postulated that one of the modifications of PrP is N-terminal truncation.

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Year:  1991        PMID: 1677605     DOI: 10.1016/0006-8993(91)91306-l

Source DB:  PubMed          Journal:  Brain Res        ISSN: 0006-8993            Impact factor:   3.252


  20 in total

1.  The primary structure of the prion protein influences the distribution of abnormal prion protein in the central nervous system.

Authors:  T Kitamoto; K Doh-ura; T Muramoto; M Miyazono; J Tateishi
Journal:  Am J Pathol       Date:  1992-08       Impact factor: 4.307

2.  Temporal evolution of sporadic Creutzfeldt-Jakob disease monitored by 3-Tesla MR spectroscopy.

Authors:  Koji Fujita; Masafumi Harada; Tatsuhiko Yuasa; Makoto Sasaki; Yuishin Izumi; Ryuji Kaji
Journal:  J Neurol       Date:  2011-02-17       Impact factor: 4.849

3.  Abnormal isoform of prion protein accumulates in follicular dendritic cells in mice with Creutzfeldt-Jakob disease.

Authors:  T Kitamoto; T Muramoto; S Mohri; K Doh-Ura; J Tateishi
Journal:  J Virol       Date:  1991-11       Impact factor: 5.103

4.  Experimental verification of a traceback phenomenon in prion infection.

Authors:  Atsushi Kobayashi; Nobuyuki Sakuma; Yuichi Matsuura; Shirou Mohri; Adriano Aguzzi; Tetsuyuki Kitamoto
Journal:  J Virol       Date:  2010-01-20       Impact factor: 5.103

5.  Species barrier prevents an abnormal isoform of prion protein from accumulating in follicular dendritic cells of mice with Creutzfeldt-Jakob disease.

Authors:  T Muramoto; T Kitamoto; M Z Hoque; J Tateishi; I Goto
Journal:  J Virol       Date:  1993-11       Impact factor: 5.103

6.  Abnormal isoform of prion proteins accumulates in the synaptic structures of the central nervous system in patients with Creutzfeldt-Jakob disease.

Authors:  T Kitamoto; R W Shin; K Doh-ura; N Tomokane; M Miyazono; T Muramoto; J Tateishi
Journal:  Am J Pathol       Date:  1992-06       Impact factor: 4.307

7.  Prion protein preamyloid and amyloid deposits in Gerstmann-Sträussler-Scheinker disease, Indiana kindred.

Authors:  G Giaccone; L Verga; O Bugiani; B Frangione; D Serban; S B Prusiner; M R Farlow; B Ghetti; F Tagliavini
Journal:  Proc Natl Acad Sci U S A       Date:  1992-10-01       Impact factor: 11.205

8.  Creutzfeldt-Jakob disease with codon 129 polymorphism (valine): a comparative study of patients with codon 102 point mutation or without mutations.

Authors:  M Miyazono; T Kitamoto; K Doh-ura; T Iwaki; J Tateishi
Journal:  Acta Neuropathol       Date:  1992       Impact factor: 17.088

9.  Immunohistochemical study of kuru plaques using antibodies against synthetic prion protein peptides.

Authors:  K Hashimoto; T Mannen; N Nukina
Journal:  Acta Neuropathol       Date:  1992       Impact factor: 17.088

10.  The coexistence of Alzheimer's disease and Creutzfeldt-Jakob disease in a patient with dementia of long duration.

Authors:  T Muramoto; T Kitamoto; H Koga; J Tateishi
Journal:  Acta Neuropathol       Date:  1992       Impact factor: 17.088

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