Literature DB >> 16771172

Effects of cotransfection of antisense-EGFR and wild-type PTEN cDNA on human glioblastoma cells.

Xin-Xia Tian1, Yun-Gang Zhang, Juan Du, Wei-Gang Fang, Ho-Keung Ng, Jie Zheng.   

Abstract

The main molecular genetic changes identified in glioblastomas are overexpression/amplification of the epidermal growth factor receptor (EGFR) gene and mutation/ deletion of the tumor suppressor PTEN gene. These two genetic changes both play important roles in glial tumorigenesis and progression. In this study, we demonstrated that wild-type PTEN transfection inhibited the growth and transforming ability of U87MG cells by 69.3% and 73.5%, respectively. On the other hand, antisense-EGFR transfection inhibited the growth and transforming phenotype of these cells by 50.3% and 46.8%, respectively. However, cotransfection of U87MG cells with wild-type PTEN and antisense EGFR constructs could inhibit the cellular growth by 91.7%. The transforming phenotype of these cells was completely inhibited. In addition, these cotransfected cells showed a differentiated form and expressed much lower telomerase activity than cells transfected with wild-type PTEN or antisense-EGFR alone. In summary, these results suggest that cotransfection is a better approach to suppress glioma cell growth than wild-type PTEN transfer or antisense-EGFR transfection alone. This approach may prove useful as an adjunct therapy in the treatment of glioblastomas.

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Year:  2006        PMID: 16771172     DOI: 10.1111/j.1440-1789.2006.00679.x

Source DB:  PubMed          Journal:  Neuropathology        ISSN: 0919-6544            Impact factor:   1.906


  7 in total

1.  Receptor activation and inhibition in cellular response to chemotherapeutic combinational mimicries: the concept of divergent targeting.

Authors:  H L Watt; Z Rachid; B J Jean-Claude
Journal:  J Neurooncol       Date:  2010-05-14       Impact factor: 4.130

2.  Combination gene therapy with PTEN and EGFR siRNA suppresses U251 malignant glioma cell growth in vitro and in vivo.

Authors:  Lei Han; An-ling Zhang; Peng Xu; Xiao Yue; Yang Yang; Guang-xiu Wang; Zhi-fan Jia; Pei-yu Pu; Chun-sheng Kang
Journal:  Med Oncol       Date:  2009-08-29       Impact factor: 3.064

Review 3.  Interactions between PTEN and receptor tyrosine kinase pathways and their implications for glioma therapy.

Authors:  Roger Abounader
Journal:  Expert Rev Anticancer Ther       Date:  2009-02       Impact factor: 4.512

4.  PTEN and Ki67 expression is associated with clinicopathologic features of non-small cell lung cancer.

Authors:  Yong Ji; Mingfeng Zheng; Shugao Ye; Jingyu Chen; Yijiang Chen
Journal:  J Biomed Res       Date:  2013-12-25

Review 5.  PI3K/Akt/mTOR signaling pathway and targeted therapy for glioblastoma.

Authors:  Xiaoman Li; Changjing Wu; Nianci Chen; Huadi Gu; Allen Yen; Liu Cao; Enhua Wang; Liang Wang
Journal:  Oncotarget       Date:  2016-05-31

6.  The drug-resistance to gefitinib in PTEN low expression cancer cells is reversed by irradiation in vitro.

Authors:  Hong-Qing Zhuang; Jun Wang; Zhi-Yong Yuan; Lu-Jun Zhao; Ping Wang; Chang-Li Wang
Journal:  J Exp Clin Cancer Res       Date:  2009-09-01

Review 7.  Targeting RTK-PI3K-mTOR Axis in Gliomas: An Update.

Authors:  Mayra Colardo; Marco Segatto; Sabrina Di Bartolomeo
Journal:  Int J Mol Sci       Date:  2021-05-05       Impact factor: 5.923

  7 in total

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