Literature DB >> 16762802

"How do cardiomyocytes die?" apoptosis and autophagic cell death in cardiac myocytes.

Sanjay Kunapuli1, Salvatore Rosanio, Ernst R Schwarz.   

Abstract

BACKGROUND: Cell death constitutes one of the key events in biology. Historically, apoptosis and necrosis have been considered to represent the 2 fundamental forms of cell death. Apoptosis is a tightly regulated, energy-dependent process in which cell death follows a programmed set of events. Necrosis refers to the sum of degenerative changes that follow any type of cell death. METHODS AND
RESULTS: The role of apoptosis in development of ischemic heart disease, hypertensive heart disease, and end-stage heart failure has been well documented. Recent evidence suggests the potential role of a third mechanism of cell death, autophagy, in loss of cardiac myocytes. Autophagic cell death has been recently documented in myocardial cells from hypertrophied, failing, and hibernating myocardium.
CONCLUSION: In this review, we will list the basic mechanisms of apoptosis and autophagic cell death and examine the recent developments in apoptosis and autophagic cell death as it pertains to cardiovascular disease.

Entities:  

Mesh:

Year:  2006        PMID: 16762802     DOI: 10.1016/j.cardfail.2006.02.002

Source DB:  PubMed          Journal:  J Card Fail        ISSN: 1071-9164            Impact factor:   5.712


  23 in total

Review 1.  Ultrastructural definition of apoptosis in heart failure.

Authors:  Eloisa Arbustini; Agnese Brega; Jagat Narula
Journal:  Heart Fail Rev       Date:  2008-06       Impact factor: 4.214

Review 2.  How does the heart (not) die? The role of autophagy in cardiomyocyte homeostasis and cell death.

Authors:  Pavittarpaul Dhesi; Faramarz Tehrani; Justin Fuess; Ernst R Schwarz
Journal:  Heart Fail Rev       Date:  2010-01       Impact factor: 4.214

3.  Serum Exosomes Attenuate H2O2-Induced Apoptosis in Rat H9C2 Cardiomyocytes via ERK1/2.

Authors:  Pengfei Li; Zhuyuan Liu; Yuan Xie; Huanyu Gu; Qiying Dai; Jianhua Yao; Lei Zhou
Journal:  J Cardiovasc Transl Res       Date:  2018-02-05       Impact factor: 4.132

4.  In vivo administration of calpeptin attenuates calpain activation and cardiomyocyte loss in pressure-overloaded feline myocardium.

Authors:  Santhosh K Mani; Hirokazu Shiraishi; Sundaravadivel Balasubramanian; Kentaro Yamane; Meenakshi Chellaiah; George Cooper; Naren Banik; Michael R Zile; Dhandapani Kuppuswamy
Journal:  Am J Physiol Heart Circ Physiol       Date:  2008-05-16       Impact factor: 4.733

5.  FoxO transcription factors promote autophagy in cardiomyocytes.

Authors:  Arunima Sengupta; Jeffery D Molkentin; Katherine E Yutzey
Journal:  J Biol Chem       Date:  2009-08-19       Impact factor: 5.157

6.  MMP9 inhibition increases autophagic flux in chronic heart failure.

Authors:  Shyam S Nandi; Kenichi Katsurada; Neeru M Sharma; Daniel R Anderson; Sushil K Mahata; Kaushik P Patel
Journal:  Am J Physiol Heart Circ Physiol       Date:  2020-10-16       Impact factor: 4.733

Review 7.  In situ ligation: a decade and a half of experience.

Authors:  Peter J Hornsby; Vladimir V Didenko
Journal:  Methods Mol Biol       Date:  2011

Review 8.  Mitochondria in heart failure: the emerging role of mitochondrial dynamics.

Authors:  José Marín-García; Alexander T Akhmedov; Gordon W Moe
Journal:  Heart Fail Rev       Date:  2013-07       Impact factor: 4.214

9.  Myocardial uptake of 99mTc-annexin-V and 111In-antimyosin-antibodies after ischemia-reperfusion in rats.

Authors:  Laure Sarda-Mantel; Florence Hervatin; Jean-Baptiste Michel; Liliane Louedec; Geneviève Martet; François Rouzet; Rachida Lebtahi; Pascal Merlet; Ban-An Khaw; Dominique Le Guludec
Journal:  Eur J Nucl Med Mol Imaging       Date:  2007-09-05       Impact factor: 9.236

10.  The involvement of lysosomes in myocardial aging and disease.

Authors:  Alexei Terman; Tino Kurz; Bertil Gustafsson; Ulf T Brunk
Journal:  Curr Cardiol Rev       Date:  2008-05
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