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6 in total

1. Complement neoantigen and vitronectin are components of plaques in amyloid AL neuropathy.

Authors: G L Zanusso; G Moretto; B Bonetti; S Monaco; N Rizzuto
Journal: Ital J Neurol Sci Date: 1992-09

2. The cerebrospinal-fluid soluble form of Alzheimer's amyloid beta is complexed to SP-40,40 (apolipoprotein J), an inhibitor of the complement membrane-attack complex.

Authors: J Ghiso; E Matsubara; A Koudinov; N H Choi-Miura; M Tomita; T Wisniewski; B Frangione
Journal: Biochem J Date: 1993-07-01 Impact factor: 3.857

3. Vascular variant of prion protein cerebral amyloidosis with tau-positive neurofibrillary tangles: the phenotype of the stop codon 145 mutation in PRNP.

Authors: B Ghetti; P Piccardo; M G Spillantini; Y Ichimiya; M Porro; F Perini; T Kitamoto; J Tateishi; C Seiler; B Frangione; O Bugiani; G Giaccone; F Prelli; M Goedert; S R Dlouhy; F Tagliavini
Journal: Proc Natl Acad Sci U S A Date: 1996-01-23 Impact factor: 11.205

Alzheimer's disease from the perspective of the systemic and localized forms of amyloidosis.

1. Complement neoantigen and vitronectin are components of plaques in amyloid AL neuropathy.

2. The cerebrospinal-fluid soluble form of Alzheimer's amyloid beta is complexed to SP-40,40 (apolipoprotein J), an inhibitor of the complement membrane-attack complex.

3. Vascular variant of prion protein cerebral amyloidosis with tau-positive neurofibrillary tangles: the phenotype of the stop codon 145 mutation in PRNP.

4. Amyloid beta 1-42 deposits do not lead to Alzheimer's neuritic plaques in aged dogs.

Review 5. Molecular biology of Alzheimer's amyloid--Dutch variant.

6. Apolipoprotein J and Alzheimer's amyloid beta solubility.