AIM: The aim was to compare the prevalence of periodontal conditions in patients with juvenile idiopathic arthritis (JIA) (n=78, age 14.4 years) with those revealed in a healthy control group (n=75, age 15.5 years). MATERIAL AND METHODS: In both groups, the approximal plaque index (API), the modified sulcular bleeding index (SBI), and the clinical attachment loss (CAL) were determined. Laboratory parameters for JIA activity included the capsule-reactive protein (CRP) and the immunoglobulins A, G, M. RESULTS: JIA patients had a significantly higher API (64.6%versus 49.9%, p=0.004) and slightly higher mean percentages of sites with CAL>3.5 mm (0.58%versus 0.22%, p=0.041). There was no significant difference in the prevalence of patients and controls who had sites with CAL >3.5 mm (25.6%versus 17.3%, p=0.212). The mean CAL was slightly greater (0.2 mm; p=0.030) in patients with CRP> or =5.0 mg/l compared with patients with CRP<5.0 mg/l. Patients who took non-steroidal anti-inflammatory drugs (NSAIDs) had a significantly decreased SBI (26.2%versus 51.1%, p=0.019). CONCLUSION: After adjustment for microbial plaque, JIA is not a risk factor for periodontitis.
AIM: The aim was to compare the prevalence of periodontal conditions in patients with juvenile idiopathic arthritis (JIA) (n=78, age 14.4 years) with those revealed in a healthy control group (n=75, age 15.5 years). MATERIAL AND METHODS: In both groups, the approximal plaque index (API), the modified sulcular bleeding index (SBI), and the clinical attachment loss (CAL) were determined. Laboratory parameters for JIA activity included the capsule-reactive protein (CRP) and the immunoglobulins A, G, M. RESULTS: JIA patients had a significantly higher API (64.6%versus 49.9%, p=0.004) and slightly higher mean percentages of sites with CAL>3.5 mm (0.58%versus 0.22%, p=0.041). There was no significant difference in the prevalence of patients and controls who had sites with CAL >3.5 mm (25.6%versus 17.3%, p=0.212). The mean CAL was slightly greater (0.2 mm; p=0.030) in patients with CRP> or =5.0 mg/l compared with patients with CRP<5.0 mg/l. Patients who took non-steroidal anti-inflammatory drugs (NSAIDs) had a significantly decreased SBI (26.2%versus 51.1%, p=0.019). CONCLUSION: After adjustment for microbial plaque, JIA is not a risk factor for periodontitis.
Authors: Lauren Lange; Geoffrey M Thiele; Courtney McCracken; Gabriel Wang; Lori A Ponder; Sheila T Angeles-Han; Kelly A Rouster-Stevens; Aimee O Hersh; Larry B Vogler; John F Bohnsack; Shelly Abramowicz; Ted R Mikuls; Sampath Prahalad Journal: Pediatr Rheumatol Online J Date: 2016-02-09 Impact factor: 3.054
Authors: Marit S Skeie; Elisabeth G Gil; Lena Cetrelli; Annika Rosén; Johannes Fischer; Anne Nordrehaug Åstrøm; Keijo Luukko; Xieqi Shi; Astrid J Feuerherm; Abhijit Sen; Paula Frid; Marite Rygg; Athanasia Bletsa Journal: BMC Oral Health Date: 2019-12-19 Impact factor: 2.757
Authors: Paula Frid; Divyashri Baraniya; Josefine Halbig; Veronika Rypdal; Nils Thomas Songstad; Annika Rosèn; Johanna Rykke Berstad; Berit Flatø; Fadhl Alakwaa; Elisabeth Grut Gil; Lena Cetrelli; Tsute Chen; Nezar Noor Al-Hebshi; Ellen Nordal; Mohammed Al-Haroni Journal: Front Cell Infect Microbiol Date: 2020-11-04 Impact factor: 5.293