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Literature DB >> 16606822

Multiple sclerosis may disrupt endocannabinoid brain protection mechanism.

Esther Shohami¹, Raphael Mechoulam.

Abstract

Entities: Disease

Mesh：

Substances：

Year: 2006 PMID： 16606822 PMCID： PMC1458835 DOI： 10.1073/pnas.0601847103

Source DB: PubMed Journal: Proc Natl Acad Sci U S A ISSN： 0027-8424 Impact factor: 11.205

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21 in total

1. Changes in cannabinoid CB(1) receptors in striatal and cortical regions of rats with experimental allergic encephalomyelitis, an animal model of multiple sclerosis.

Authors: F Berrendero; A Sánchez; A Cabranes; C Puerta; J A Ramos; A García-Merino; J Fernández-Ruiz
Journal: Synapse Date: 2001-09-01 Impact factor: 2.562

Review 2. New perspectives in the studies on endocannabinoid and cannabis: abnormal behaviors associate with CB1 cannabinoid receptor and development of therapeutic application.

Authors: Michihiro Fujiwara; Nobuaki Egashira
Journal: J Pharmacol Sci Date: 2004-12-10 Impact factor: 3.337

3. CB1 cannabinoid receptors are involved in neuroprotection via NF-kappa B inhibition.

Authors: David Panikashvili; Raphael Mechoulam; Sara M Beni; Alexander Alexandrovich; Esther Shohami
Journal: J Cereb Blood Flow Metab Date: 2005-04 Impact factor: 6.200

4. Cannabinoids and neuroprotection in global and focal cerebral ischemia and in neuronal cultures.

Authors: T Nagayama; A D Sinor; R P Simon; J Chen; S H Graham; K Jin; D A Greenberg
Journal: J Neurosci Date: 1999-04-15 Impact factor: 6.167

5. An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

Authors: D Panikashvili; C Simeonidou; S Ben-Shabat; L Hanus; A Breuer; R Mechoulam; E Shohami
Journal: Nature Date: 2001-10-04 Impact factor: 49.962

6. Treatment with soluble tumor necrosis factor receptor (sTNFR):Fc/p80 fusion protein ameliorates relapsing-remitting experimental autoimmune encephalomyelitis and decreases chemokine expression.

Authors: Andrzej R Glabinski; Bartosz Bielecki; Julie A Kawczak; Vincent K Tuohy; Krzysztof Selmaj; Richard M Ransohoff
Journal: Autoimmunity Date: 2004 Sep-Nov Impact factor: 2.815

Multiple sclerosis may disrupt endocannabinoid brain protection mechanism.

1. Changes in cannabinoid CB(1) receptors in striatal and cortical regions of rats with experimental allergic encephalomyelitis, an animal model of multiple sclerosis.

Review 2. New perspectives in the studies on endocannabinoid and cannabis: abnormal behaviors associate with CB1 cannabinoid receptor and development of therapeutic application.

3. CB1 cannabinoid receptors are involved in neuroprotection via NF-kappa B inhibition.

4. Cannabinoids and neuroprotection in global and focal cerebral ischemia and in neuronal cultures.

5. An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

6. Treatment with soluble tumor necrosis factor receptor (sTNFR):Fc/p80 fusion protein ameliorates relapsing-remitting experimental autoimmune encephalomyelitis and decreases chemokine expression.

7. Suppression of experimental autoimmune encephalomyelitis by cannabinoids.

8. Identification of an endogenous 2-monoglyceride, present in canine gut, that binds to cannabinoid receptors.

9. Cannabinoid-receptor 1 null mice are susceptible to neurofilament damage and caspase 3 activation.

10. Cannabinoids control spasticity and tremor in a multiple sclerosis model.

Review 1. Cardiovascular Autonomic Dysfunction: Link Between Multiple Sclerosis Osteoporosis and Neurodegeneration.

2. Endocannabinoid signaling in midbrain dopamine neurons: more than physiology?