Literature DB >> 1660249

Two high-threshold Ca2+ channels contribute Ca2+ for depolarization-secretion coupling in the mammalian neurohypophysis.

M C Nowycky1.   

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Year:  1991        PMID： 1660249     DOI： 10.1111/j.1749-6632.1991.tb36480.x

Source DB:  PubMed          Journal:  Ann N Y Acad Sci        ISSN： 0077-8923            Impact factor:   5.691


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  5 in total

1.  Ca2+- and voltage-dependent inactivation of Ca2+ channels in nerve terminals of the neurohypophysis.

Authors:  J L Branchaw; M I Banks; M B Jackson
Journal:  J Neurosci       Date:  1997-08-01       Impact factor: 6.167

Review 2.  Mechanisms in the regulation of neurotransmitter release from brain nerve terminals: current hypotheses.

Authors:  T S Sihra; R A Nichols
Journal:  Neurochem Res       Date:  1993-01       Impact factor: 3.996

3.  Excitation-secretion coupling in mammalian neurohypophysial nerve terminals.

Authors:  M C Nowycky; E P Seward; N I Chernevskaya
Journal:  Cell Mol Neurobiol       Date:  1998-02       Impact factor: 5.046

4.  Distinct omega-agatoxin-sensitive calcium currents in somata and axon terminals of rat supraoptic neurones.

Authors:  T E Fisher; C W Bourque
Journal:  J Physiol       Date:  1995-12-01       Impact factor: 5.182

5.  Muscarinic (M1) receptor-mediated inhibition of K(+)-evoked [3H]-noradrenaline release from human neuroblastoma (SH-SY5Y) cells via inhibition of L- and N-type Ca2+ channels.

Authors:  R L McDonald; P F Vaughan; C Peers
Journal:  Br J Pharmacol       Date:  1994-10       Impact factor: 8.739
  5 in total

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