Literature DB >> 16508957

Comparative analysis of gene expression profiles in intact and damaged regions of human osteoarthritic cartilage.

Tomoo Sato1, Koji Konomi, Satoshi Yamasaki, Satoko Aratani, Kaneyuki Tsuchimochi, Masahiro Yokouchi, Kayo Masuko-Hongo, Naoko Yagishita, Hiroshi Nakamura, Setsuro Komiya, Moroe Beppu, Haruhito Aoki, Kusuki Nishioka, Toshihiro Nakajima.   

Abstract

OBJECTIVE: To analyze the differences in gene expression profiles of chondrocytes in intact and damaged regions of cartilage from the same knee joint of patients with osteoarthritis (OA) of the knee.
METHODS: We compared messenger RNA expression profiles in regions of intact and damaged cartilage (classified according to the Mankin scale) obtained from patients with knee OA. Five pairs of intact and damaged regions of OA cartilage were evaluated by oligonucleotide array analysis using a double in vitro transcription amplification technique. The microarray data were confirmed by real-time quantitative polymerase chain reaction (PCR) amplification and were compared with previously published data.
RESULTS: About 1,500 transcripts, which corresponded to 8% of the expressed transcripts, showed > or = 2-fold differences in expression between the cartilage tissue pairs. Approximately 10% of these transcripts (n = 151) were commonly expressed in the 5 patient samples. Accordingly, 114 genes (35 genes expressed in intact > damaged; 79 genes expressed in intact < damaged) were selected. The expression of some genes related to the wound-healing process, including cell proliferation and interstitial collagen synthesis, was higher in damaged regions than in intact regions, similar to the findings for genes that inhibit matrix degradation. Comparisons of the real-time quantitative PCR data with the previously reported data support the validity of our microarray data.
CONCLUSION: Differences between intact and damaged regions of OA cartilage exhibited a similar pattern among the 5 patients examined, indicating the presence of common mechanisms that contribute to cartilage destruction. Elucidation of this mechanism is important for the development of effective treatments for OA.

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Year:  2006        PMID: 16508957     DOI: 10.1002/art.21638

Source DB:  PubMed          Journal:  Arthritis Rheum        ISSN: 0004-3591


  61 in total

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4.  Telomerase transduced osteoarthritis fibroblast-like synoviocytes display a distinct gene expression profile.

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Review 6.  Nutritional, metabolic and genetic considerations to optimise regenerative medicine outcome for knee osteoarthritis.

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7.  Phenotypic characterization of epiphycan-deficient and epiphycan/biglycan double-deficient mice.

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8.  Differential expression of GADD45beta in normal and osteoarthritic cartilage: potential role in homeostasis of articular chondrocytes.

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9.  DNA demethylation at specific CpG sites in the IL1B promoter in response to inflammatory cytokines in human articular chondrocytes.

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Review 10.  Cartilage homeostasis in health and rheumatic diseases.

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