Literature DB >> 16461566

Specific epitopes of the structural and hypothetical proteins elicit variable humoral responses in SARS patients.

S C S Chow1, C Y S Ho, T T Y Tam, C Wu, T Cheung, P K S Chan, M H L Ng, P K Hui, H K Ng, D M Y Au, A W I Lo.   

Abstract

BACKGROUND: Severe acute respiratory syndrome (SARS) is an infectious disease which was caused by a novel coronavirus (SARS-CoV). SARS has caused an outbreak in the world during 2003 and 2004, with 8098 individuals being infected and a death toll of 774 in 28 regions around the world. Specific humoral responses to viral infection remain unclear.
OBJECTIVE: To analyse the antigenicity of the SARS-CoV genome and identify potential antigenic epitopes in the structural proteins.
METHODS: Potential antigenic epitopes were identified in the structural proteins (nucleocapsid, membrane, spike, and small envelope proteins) and hypothetical proteins (SARS3a, 3b, 6, 7a, and 9b) that are specific for SARS-CoV. A peptide chip platform was created and the profiles of antibodies to these epitopes were investigated in 59 different SARS patients' sera obtained 6-103 days after the onset of the illness. Serial sera from five additional patients were also studied.
RESULTS: Epitopes at the N-terminus of the membrane protein and the C-terminus of nucleocapsid protein elicited strong antibody responses. Epitopes on the spike protein were only moderately immunogenic but the effects were persistent. Antibodies were also detected for some putative proteins, noticeably the C-termini of SARS3a and SARS6.
CONCLUSIONS: Important epitopes of the SARS-CoV genome that may serve as potential markers for the viral infection are identified. These specific antigenic sites may also be important for vaccine development against this new fatal infectious disease.

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Year:  2006        PMID: 16461566      PMCID: PMC1860290          DOI: 10.1136/jcp.2005.029868

Source DB:  PubMed          Journal:  J Clin Pathol        ISSN: 0021-9746            Impact factor:   3.411


  22 in total

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Journal:  Science       Date:  2003-05-01       Impact factor: 47.728

2.  The Genome sequence of the SARS-associated coronavirus.

Authors:  Marco A Marra; Steven J M Jones; Caroline R Astell; Robert A Holt; Angela Brooks-Wilson; Yaron S N Butterfield; Jaswinder Khattra; Jennifer K Asano; Sarah A Barber; Susanna Y Chan; Alison Cloutier; Shaun M Coughlin; Doug Freeman; Noreen Girn; Obi L Griffith; Stephen R Leach; Michael Mayo; Helen McDonald; Stephen B Montgomery; Pawan K Pandoh; Anca S Petrescu; A Gordon Robertson; Jacqueline E Schein; Asim Siddiqui; Duane E Smailus; Jeff M Stott; George S Yang; Francis Plummer; Anton Andonov; Harvey Artsob; Nathalie Bastien; Kathy Bernard; Timothy F Booth; Donnie Bowness; Martin Czub; Michael Drebot; Lisa Fernando; Ramon Flick; Michael Garbutt; Michael Gray; Allen Grolla; Steven Jones; Heinz Feldmann; Adrienne Meyers; Amin Kabani; Yan Li; Susan Normand; Ute Stroher; Graham A Tipples; Shaun Tyler; Robert Vogrig; Diane Ward; Brynn Watson; Robert C Brunham; Mel Krajden; Martin Petric; Danuta M Skowronski; Chris Upton; Rachel L Roper
Journal:  Science       Date:  2003-05-01       Impact factor: 47.728

3.  Diagnosis of severe acute respiratory syndrome (SARS) by detection of SARS coronavirus nucleocapsid antibodies in an antigen-capturing enzyme-linked immunosorbent assay.

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Journal:  J Clin Pathol       Date:  2004-03       Impact factor: 3.411

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Journal:  Nature       Date:  2003-11-27       Impact factor: 49.962

10.  A 193-amino acid fragment of the SARS coronavirus S protein efficiently binds angiotensin-converting enzyme 2.

Authors:  Swee Kee Wong; Wenhui Li; Michael J Moore; Hyeryun Choe; Michael Farzan
Journal:  J Biol Chem       Date:  2003-12-11       Impact factor: 5.157

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5.  De novo design of modular and tunable protein biosensors.

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Review 7.  Humoral Immune Responses in COVID-19 Patients: A Window on the State of the Art.

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Review 9.  The role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesis.

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10.  Analysis of intraviral protein-protein interactions of the SARS coronavirus ORFeome.

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