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Literature DB >> 16458005

Chromatin regulation of virus infection.

Abstract

Cellular chromatin forms a dynamic structure that maintains the stability and accessibility of the host DNA genome. Viruses that enter and persist in the nucleus must, therefore, contend with the forces that drive chromatin formation and regulate chromatin structure. In some cases, cellular chromatin inhibits viral gene expression and replication by suppressing DNA accessibility. In other cases, cellular chromatin provides essential structure and organization to the viral genome and is necessary for successful completion of the viral life cycle. Consequently, viruses have acquired numerous mechanisms to manipulate cellular chromatin to ensure viral genome survival and propagation.

Entities: Disease

Mesh：

Substances：
Chromatin

Year: 2006 PMID： 16458005 DOI： 10.1016/j.tim.2006.01.001

Source DB: PubMed Journal: Trends Microbiol ISSN： 0966-842X Impact factor: 17.079

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Cited

38 in total

1. Promoter-Targeted Histone Acetylation of Chromatinized Parvoviral Genome Is Essential for the Progress of Infection.

Authors: Elina Mäntylä; Kari Salokas; Mikko Oittinen; Vesa Aho; Pekka Mäntysaari; Lassi Palmujoki; Olli Kalliolinna; Teemu O Ihalainen; Einari A Niskanen; Jussi Timonen; Keijo Viiri; Maija Vihinen-Ranta
Journal: J Virol Date: 2016-03-28 Impact factor: 5.103

2. The protamine-like DNA-binding protein P6.9 epigenetically up-regulates Autographa californica multiple nucleopolyhedrovirus gene transcription in the late infection phase.

Authors: Ying Peng; Kun Li; Rong-juan Pei; Chun-chen Wu; Chang-yong Liang; Yun Wang; Xin-wen Chen
Journal: Virol Sin Date: 2012-01-22 Impact factor: 4.327

3. Histone modifications induced by a family of bacterial toxins.

Authors: Mélanie Anne Hamon; Eric Batsché; Béatrice Régnault; To Nam Tham; Stéphanie Seveau; Christian Muchardt; Pascale Cossart
Journal: Proc Natl Acad Sci U S A Date: 2007-08-03 Impact factor: 11.205

4. High-frequency epigenetic repression and silencing of retroviruses can be antagonized by histone deacetylase inhibitors and transcriptional activators, but uniform reactivation in cell clones is restricted by additional mechanisms.

Authors: Richard A Katz; Emily Jack-Scott; Anna Narezkina; Ivan Palagin; Pamela Boimel; Joseph Kulkosky; Emmanuelle Nicolas; James G Greger; Anna Marie Skalka
Journal: J Virol Date: 2007-01-03 Impact factor: 5.103

5. Identification of cellular proteins that maintain retroviral epigenetic silencing: evidence for an antiviral response.

Authors: Andrey Poleshko; Ivan Palagin; Rugang Zhang; Pamela Boimel; Carolyn Castagna; Peter D Adams; Anna Marie Skalka; Richard A Katz
Journal: J Virol Date: 2007-12-19 Impact factor: 5.103

Chromatin regulation of virus infection.

1. Promoter-Targeted Histone Acetylation of Chromatinized Parvoviral Genome Is Essential for the Progress of Infection.

2. The protamine-like DNA-binding protein P6.9 epigenetically up-regulates Autographa californica multiple nucleopolyhedrovirus gene transcription in the late infection phase.

3. Histone modifications induced by a family of bacterial toxins.

4. High-frequency epigenetic repression and silencing of retroviruses can be antagonized by histone deacetylase inhibitors and transcriptional activators, but uniform reactivation in cell clones is restricted by additional mechanisms.

5. Identification of cellular proteins that maintain retroviral epigenetic silencing: evidence for an antiviral response.

6. Identification of a functional network of human epigenetic silencing factors.

7. During lytic infections, herpes simplex virus type 1 DNA is in complexes with the properties of unstable nucleosomes.

8. Reactivation of DNA viruses in association with histone deacetylase inhibitor therapy: a case series report.

9. An atlas of the Epstein-Barr virus transcriptome and epigenome reveals host-virus regulatory interactions.

10. Proteomic analysis reveals selective impediment of neuronal remodeling upon Borna disease virus infection.