Literature DB >> 16435156

FK228 (depsipeptide): a HDAC inhibitor with pleiotropic antitumor activities.

Panagiotis A Konstantinopoulos1, Gerasimos P Vandoros, Athanasios G Papavassiliou.   

Abstract

PURPOSE: The fundamental role of epigenetic events in carcinogenesis has resulted in the evolution of epigenetic targeting as a new paradigm in anticancer therapeutics. Aberrant histone deacetylase (HDAC) activity has been documented in many human malignancies resulting in the repression of tumor suppressor genes and promotion of tumorigenesis. FK228, also known as depsipeptide, is a novel, natural, bicyclic tetrapeptide with significant antitumor properties which are mostly mediated by inhibition of HDACs.
RESULTS: FK228 induces the expression of genes linked to the inhibition of cell growth, induction of cell differentiation, promotion of apoptotic cell death and inhibition of angiogenesis.
CONCLUSION: Its multitargeting properties, its ability to act on non-histone targets, its clinical activity and its acceptable side-effect profile render FK228 a very promising novel anticancer agent.

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Year:  2006        PMID: 16435156     DOI: 10.1007/s00280-005-0182-5

Source DB:  PubMed          Journal:  Cancer Chemother Pharmacol        ISSN: 0344-5704            Impact factor:   3.333


  19 in total

1.  Romidepsin: in the treatment of T-cell lymphoma.

Authors:  Lily P H Yang
Journal:  Drugs       Date:  2011-07-30       Impact factor: 9.546

2.  The HDAC inhibitor FK228 enhances adenoviral transgene expression by a transduction-independent mechanism but does not increase adenovirus replication.

Authors:  Angelika Danielsson; Helena Dzojic; Victoria Rashkova; Wing-Shing Cheng; Magnus Essand
Journal:  PLoS One       Date:  2011-02-17       Impact factor: 3.240

3.  Reexpression of tumor suppressor, sFRP1, leads to antitumor synergy of combined HDAC and methyltransferase inhibitors in chemoresistant cancers.

Authors:  Simon J Cooper; Christina A von Roemeling; Kylie H Kang; Laura A Marlow; Stefan K Grebe; Michael E Menefee; Han W Tun; Gerardo Colon-Otero; Edith A Perez; John A Copland
Journal:  Mol Cancer Ther       Date:  2012-07-23       Impact factor: 6.261

4.  Histone deacetylase inhibitors in the treatment for multiple myeloma.

Authors:  Teru Hideshima; Kenneth C Anderson
Journal:  Int J Hematol       Date:  2013-03-10       Impact factor: 2.490

Review 5.  HDAC inhibitor-based therapies: can we interpret the code?

Authors:  Maria New; Heidi Olzscha; Nicholas B La Thangue
Journal:  Mol Oncol       Date:  2012-10-23       Impact factor: 6.603

Review 6.  Cutaneous T cell Lymphoma: an Update on Pathogenesis and Systemic Therapy.

Authors:  Catherine G Chung; Brian Poligone
Journal:  Curr Hematol Malig Rep       Date:  2015-12       Impact factor: 3.952

7.  Histone deacetylase inhibitor romidepsin enhances anti-tumor effect of erlotinib in non-small cell lung cancer (NSCLC) cell lines.

Authors:  Wei Zhang; Michael Peyton; Yang Xie; Junichi Soh; John D Minna; Adi F Gazdar; Eugene P Frenkel
Journal:  J Thorac Oncol       Date:  2009-02       Impact factor: 15.609

Review 8.  Novel histone deacetylase inhibitors in clinical trials as anti-cancer agents.

Authors:  Jiahuai Tan; Shundong Cang; Yuehua Ma; Richard L Petrillo; Delong Liu
Journal:  J Hematol Oncol       Date:  2010-02-04       Impact factor: 17.388

9.  Pro-apoptotic activity of oncogenic H-Ras for histone deacetylase inhibitor to induce apoptosis of human cancer HT29 cells.

Authors:  Shambhunath Choudhary; Hwa-Chain Robert Wang
Journal:  J Cancer Res Clin Oncol       Date:  2007-05-04       Impact factor: 4.553

10.  Romidepsin inhibits Ras-dependent growth transformation of NIH 3T3 fibroblasts and RIE-1 epithelial cells independently of Ras signaling inhibition.

Authors:  Ariella B Hanker; Kevin D Healy; Jean Nichols; Channing J Der
Journal:  J Mol Signal       Date:  2009-08-16
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