Literature DB >> 16420507

Serum cytokine and soluble cytokine receptor levels in patients with non-alcoholic steatohepatitis.

Seigo Abiru1, Kiyoshi Migita, Yumi Maeda, Manabu Daikoku, Masahiro Ito, Kazuyuki Ohata, Shinya Nagaoka, Takehiro Matsumoto, Yasushi Takii, Koichiro Kusumoto, Minoru Nakamura, Atsumasa Komori, Koji Yano, Hiroshi Yatsuhashi, Katsumi Eguchi, Hiromi Ishibashi.   

Abstract

BACKGROUND: Although the pathogenesis of non-alcoholic steatohepatitis (NASH) remains poorly understood, proinflammatory cytokines seem to play an important role in the process of NASH. We have undertaken this study in order to elucidate the role of proinflammatory cytokines and their soluble receptors in NASH patients.
METHODS: Serum cytokines and soluble cytokine receptors levels were determined using an enzyme-linked immunosorbent assay kit in 23 patients with NASH, 21 patients with simple steatosis, and 18 healthy volunteers.
RESULTS: Patients with NASH had significantly higher serum tumor necrosis factor-alpha (TNF-alpha) and interleukin-6 (IL-6) levels than did the simple steatosis patients. Similarly, when compared with simple steatosis, NASH was associated with higher soluble TNF receptor 1 (sTNFR1) and soluble IL-6 receptor (sIL-6R) levels, and a significant positive correlation was seen between the levels of sTNFR1 and aminotranferases in NASH patients.
CONCLUSIONS: This study shows that circulating TNF-alpha/sTNFR1 and IL-6/sIL-6R levels are significantly increased in NASH patients as compared with simple steatosis patients and healthy volunteers, and that these increased levels may be implicated in the pathogenesis of NASH.

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Year:  2006        PMID: 16420507     DOI: 10.1111/j.1478-3231.2005.01191.x

Source DB:  PubMed          Journal:  Liver Int        ISSN: 1478-3223            Impact factor:   5.828


  70 in total

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6.  Tissue factor-deficiency and protease activated receptor-1-deficiency reduce inflammation elicited by diet-induced steatohepatitis in mice.

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8.  Kuppfer cells trigger nonalcoholic steatohepatitis development in diet-induced mouse model through tumor necrosis factor-α production.

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Journal:  J Biol Chem       Date:  2012-10-12       Impact factor: 5.157

Review 9.  The role of hepatic fat accumulation in pathogenesis of non-alcoholic fatty liver disease (NAFLD).

Authors:  Qing Liu; Stig Bengmark; Shen Qu
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10.  IL-6 deficiency attenuates murine diet-induced non-alcoholic steatohepatitis.

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