Literature DB >> 16409462

A rapid enzyme-linked assay for ADAMTS-13.

J-J Wu1, K Fujikawa, E C Lian, B A McMullen, J D Kulman, D W Chung.   

Abstract

BACKGROUND: A deficiency in the plasma metalloprotease ADAMTS-13 is associated with deposition of microvascular thrombi that cause thrombotic thrombocytopenic purpura. Current assays for ADAMTS-13 are technically complex and time-consuming. The objective of this study is to devise a rapid and sensitive assay for ADAMTS-13 activity in plasma and verify the site of cleavage.
METHOD: A new enzyme-linked substrate, which contains a core ADAMTS-13-specific peptide conjugated to horseradish peroxidase (HRP) at the N-terminus, and labeled with biotin at the C-terminus, was constructed. After cleavage of this substrate by plasma ADAMTS-13 and removal of uncleaved substrate by adsorption with streptavidin-agarose, ADAMTS-13 activity was quantitated by determining the unadsorbed HRP activity remaining in solution. Levels of inhibitory antibodies in test plasma were also determined by measuring the residual ADAMTS-13 activity after varying amounts of test plasma were incubated with a known amount of ADAMTS-13.
RESULTS: Plasma ADAMTS-13 activity was readily determined in approximately 60 min (coefficient of variation 5.8%) using 1 microL of test plasma. Amino acid sequencing of the cleavage product confirmed that cleavage occurred at the Tyr1605-Met1606 bond in the substrate. ADAMTS-13 activities in the plasma of five TTP patients were below 2%. Inhibitory antibody titers in these samples varied from undetectable to 81 BU mL(-1).
CONCLUSION: The HRP-linked substrate provides a rapid, sensitive, and reproducible way of determining the levels of ADAMTS-13 activity and inhibitory antibodies in plasma.

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Year:  2006        PMID: 16409462     DOI: 10.1111/j.1538-7836.2005.01677.x

Source DB:  PubMed          Journal:  J Thromb Haemost        ISSN: 1538-7836            Impact factor:   5.824


  14 in total

1.  Inhibitory autoantibodies against ADAMTS-13 in patients with thrombotic thrombocytopenic purpura bind ADAMTS-13 protease and may accelerate its clearance in vivo.

Authors:  S G Shelat; P Smith; J Ai; X L Zheng
Journal:  J Thromb Haemost       Date:  2006-08       Impact factor: 5.824

2.  Measurement of ADAMTS13.

Authors:  Han-Mou Tsai
Journal:  Int Rev Thromb       Date:  2006

3.  Endothelial Activation and Blood-Brain Barrier Disruption in Neurotoxicity after Adoptive Immunotherapy with CD19 CAR-T Cells.

Authors:  Juliane Gust; Kevin A Hay; Laïla-Aïcha Hanafi; Daniel Li; David Myerson; Luis F Gonzalez-Cuyar; Cecilia Yeung; W Conrad Liles; Mark Wurfel; Jose A Lopez; Junmei Chen; Dominic Chung; Susanna Harju-Baker; Tahsin Özpolat; Kathleen R Fink; Stanley R Riddell; David G Maloney; Cameron J Turtle
Journal:  Cancer Discov       Date:  2017-10-12       Impact factor: 39.397

4.  Exosite interactions contribute to tension-induced cleavage of von Willebrand factor by the antithrombotic ADAMTS13 metalloprotease.

Authors:  Weiqiang Gao; Patricia J Anderson; Elaine M Majerus; Elodee A Tuley; J Evan Sadler
Journal:  Proc Natl Acad Sci U S A       Date:  2006-12-04       Impact factor: 11.205

5.  Rearranging exosites in noncatalytic domains can redirect the substrate specificity of ADAMTS proteases.

Authors:  Weiqiang Gao; Jian Zhu; Lisa A Westfield; Elodee A Tuley; Patricia J Anderson; J Evan Sadler
Journal:  J Biol Chem       Date:  2012-06-15       Impact factor: 5.157

6.  Hypochlorous acid generated by neutrophils inactivates ADAMTS13: an oxidative mechanism for regulating ADAMTS13 proteolytic activity during inflammation.

Authors:  Yi Wang; Junmei Chen; Minhua Ling; José A López; Dominic W Chung; Xiaoyun Fu
Journal:  J Biol Chem       Date:  2014-11-24       Impact factor: 5.157

7.  The rate of hemolysis in sickle cell disease correlates with the quantity of active von Willebrand factor in the plasma.

Authors:  Junmei Chen; William E Hobbs; Jennie Le; Peter J Lenting; Philip G de Groot; José A López
Journal:  Blood       Date:  2011-02-07       Impact factor: 22.113

Review 8.  Pathophysiology of thrombotic thrombocytopenic purpura.

Authors:  Han-Mou Tsai
Journal:  Int J Hematol       Date:  2010-01       Impact factor: 2.490

9.  Normal cleavage of von Willebrand factor by ADAMTS-13 in the absence of factor VIII in patients with severe hemophilia A.

Authors:  J Chen; D W Chung; J Le; M Ling; B A Konkle; J A López
Journal:  J Thromb Haemost       Date:  2013-09       Impact factor: 5.824

10.  Oxidative modification of von Willebrand factor by neutrophil oxidants inhibits its cleavage by ADAMTS13.

Authors:  Junmei Chen; Xiaoyun Fu; Yi Wang; Minhua Ling; Brad McMullen; John Kulman; Dominic W Chung; José A López
Journal:  Blood       Date:  2009-10-07       Impact factor: 22.113

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