Warning: Undefined array key "mm" in /www/wwwroot/www.ai-bt.com/si.php on line 10 Deprecated: trim(): Passing null to parameter #1 ($string) of type string is deprecated in /www/wwwroot/www.ai-bt.com/si.php on line 10 Unexpected role of TRPC6 channel in familial nephrotic syndrome: does it have clinical implications?

Literature DB >> 16396961

Unexpected role of TRPC6 channel in familial nephrotic syndrome: does it have clinical implications?

Michelle P Winn¹, Nikki Daskalakis, Robert F Spurney, John P Middleton.

Abstract

Entities: Disease Gene

Mesh：

Substances：

Year: 2006 PMID： 16396961 DOI： 10.1681/ASN.2005090962

Source DB: PubMed Journal: J Am Soc Nephrol ISSN： 1046-6673 Impact factor: 10.121

Keyword Cloud
Cited

× No keyword cloud information.

25 in total

Review 1. TRPC6 in glomerular health and disease: what we know and what we believe.

Authors: Johannes S Schlöndorff; Martin R Pollak
Journal: Semin Cell Dev Biol Date: 2006-11-20 Impact factor: 7.727

Review 2. Transient receptor potential channelopathies.

Authors: Bernd Nilius; Grzegorz Owsianik
Journal: Pflugers Arch Date: 2010-02-04 Impact factor: 3.657

3. Gq signaling causes glomerular injury by activating TRPC6.

Authors: Liming Wang; Grant Jirka; Paul B Rosenberg; Anne F Buckley; Jose A Gomez; Timothy A Fields; Michelle P Winn; Robert F Spurney
Journal: J Clin Invest Date: 2015-04-06 Impact factor: 14.808

4. A novel mouse model of podocyte depletion.

Authors: L Wang; Y Tang; D N Howell; P Ruiz; R F Spurney
Journal: Nephron Exp Nephrol Date: 2012-10-19

5. Gq-dependent signaling upregulates COX2 in glomerular podocytes.

Authors: Liming Wang; Patrick J Flannery; Paul B Rosenberg; Timothy A Fields; Robert F Spurney
Journal: J Am Soc Nephrol Date: 2008-07-30 Impact factor: 10.121

6. Nuclear factor κB mediates suppression of canonical transient receptor potential 6 expression by reactive oxygen species and protein kinase C in kidney cells.

Authors: Yanxia Wang; Min Ding; Sarika Chaudhari; Yanfeng Ding; Joseph Yuan; Dorota Stankowska; Shaoqing He; Raghu Krishnamoorthy; Joseph T Cunningham; Rong Ma
Journal: J Biol Chem Date: 2013-03-22 Impact factor: 5.157

Unexpected role of TRPC6 channel in familial nephrotic syndrome: does it have clinical implications?

Review 1. TRPC6 in glomerular health and disease: what we know and what we believe.

Review 2. Transient receptor potential channelopathies.

3. Gq signaling causes glomerular injury by activating TRPC6.

4. A novel mouse model of podocyte depletion.

5. Gq-dependent signaling upregulates COX2 in glomerular podocytes.

6. Nuclear factor κB mediates suppression of canonical transient receptor potential 6 expression by reactive oxygen species and protein kinase C in kidney cells.

7. Single-channel analysis of TRPC channels in the podocytes of freshly isolated Glomeruli.

Review 8. Minimal change nephropathy and focal segmental glomerulosclerosis.

9. Canonical transient receptor potential 6 (TRPC6), a redox-regulated cation channel.

10. Genetic forms of nephrotic syndrome: a single-center experience in Brussels.