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9 in total

1. Toxoplasma gondii strains defective in oral transmission are also defective in developmental stage differentiation.

Authors: Blima Fux; Julie Nawas; Asis Khan; Darcy B Gill; Chunlei Su; L David Sibley
Journal: Infect Immun Date: 2007-03-05 Impact factor: 3.441

2. Alternative splicing mechanisms orchestrating post-transcriptional gene expression: intron retention and the intron-rich genome of apicomplexan parasites.

Authors: Matteo Lunghi; Furio Spano; Alessandro Magini; Carla Emiliani; Vern B Carruthers; Manlio Di Cristina
Journal: Curr Genet Date: 2015-07-21 Impact factor: 3.886

Review 3. Observations on bradyzoite biology.

Authors: Vincent Tu; Rama Yakubu; Louis M Weiss
Journal: Microbes Infect Date: 2017-12-26 Impact factor: 2.700

4. Type II Toxoplasma gondii KU80 knockout strains enable functional analysis of genes required for cyst development and latent infection.

Authors: Barbara A Fox; Alejandra Falla; Leah M Rommereim; Tadakimi Tomita; Jason P Gigley; Corinne Mercier; Marie-France Cesbron-Delauw; Louis M Weiss; David J Bzik
Journal: Eukaryot Cell Date: 2011-04-29

Disruption of the bradyzoite-specific P-type (H+)-ATPase PMA1 in Toxoplasma gondii leads to decreased bradyzoite differentiation after stress stimuli but does not interfere with mature tissue cyst formation.

1. Toxoplasma gondii strains defective in oral transmission are also defective in developmental stage differentiation.

2. Alternative splicing mechanisms orchestrating post-transcriptional gene expression: intron retention and the intron-rich genome of apicomplexan parasites.

Review 3. Observations on bradyzoite biology.

4. Type II Toxoplasma gondii KU80 knockout strains enable functional analysis of genes required for cyst development and latent infection.

Review 5. Mechanisms of Toxoplasma gondii persistence and latency.

6. A pseudouridine synthase homologue is critical to cellular differentiation in Toxoplasma gondii.

Review 7. A latent ability to persist: differentiation in Toxoplasma gondii.

8. Deletion of mitogen-activated protein kinase 1 inhibits development and growth of Toxoplasma gondii.

9. The GRA17 Parasitophorous Vacuole Membrane Permeability Pore Contributes to Bradyzoite Viability.