Literature DB >> 16371896

The TRP2 allele of COL9A2 is an age-dependent risk factor for the development and severity of intervertebral disc degeneration.

Jeffrey J T Jim1, Noora Noponen-Hietala, Kenneth M C Cheung, Jürg Ott, Jaro Karppinen, Ahmad Sahraravand, Keith D K Luk, Shea-Ping Yip, Pak C Sham, You-Qiang Song, John C Y Leong, Kathryn S E Cheah, Leena Ala-Kokko, Danny Chan.   

Abstract

STUDY
DESIGN: Low back pain (LBP) and sciatica are usually caused by degenerative disc disease (DDD). Although they are common, the etiology of these conditions is poorly understood. A large population case-control study in the Southern Chinese was performed to study genetic risk factors to DDD.
OBJECTIVES: To gain a better understanding of the etiology of DDD in relation to structural defects of the intervertebral disc. SUMMARY OF BACKGROUND DATA: A Finnish study found an association between LBP and sciatica with two variants of the alpha-chains of collagen IX, encoded by the Trp2 and Trp3 alleles, representing Gln326Trp and Arg103Trp amino acid substitutions in the COL9A2 and COL9A3 genes, respectively. Trp2 was found only in affected individuals (4%), whereas Trp3 was present in both affected (24%) and unaffected (9%) individuals. Because of the low frequency of the Trp2 allele in whites, the significance and contribution of this allele to DDD are not known. Using more objective criteria to define the disease by magnetic resonance imaging (MRI), we tested these alleles for association with DDD in a large population study.
METHODS: Lumbar DDD, the presence of anular tears, and disc and endplate herniations were defined by MRI in 804 Southern Chinese volunteers 18 to 55 years of age. These were correlated with the frequencies of the Trp2 and Trp3 alleles.
RESULTS: The Trp2 allele was present in 20% of the population and was associated with a fourfold increase in the risk of developing anular tears at 30 to 39 years and a 2.4-fold increase in the risk of developing DDD and endplate herniations at 40 to 49 years. Affected Trp2 individuals had more severe degeneration. The Trp3 allele was absent from the Southern Chinese population.
CONCLUSION: This largest-ever population study using MRI to define DDD demonstrates for the first time that the Trp2 allele is a significant risk factor for the development and severity of degeneration. The association is age- dependent as it is more prevalent in some age groups than in others. The contrasting Trp allele frequencies between the Finns and the Chinese are the first indication that the genetic risk factors for DDD varies between ethnic groups.

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Year:  2005        PMID: 16371896     DOI: 10.1097/01.brs.0000190828.85331.ef

Source DB:  PubMed          Journal:  Spine (Phila Pa 1976)        ISSN: 0362-2436            Impact factor:   3.468


  40 in total

1.  The postnatal role of Sox9 in cartilage.

Authors:  Stephen P Henry; Shoudan Liang; Kadir C Akdemir; Benoit de Crombrugghe
Journal:  J Bone Miner Res       Date:  2012-12       Impact factor: 6.741

Review 2.  Regeneration of intervertebral disc by mesenchymal stem cells: potentials, limitations, and future direction.

Authors:  Victor Y L Leung; Danny Chan; Kenneth M C Cheung
Journal:  Eur Spine J       Date:  2006-07-15       Impact factor: 3.134

3.  Degenerative disc disease: genotyping, MR imaging and phenotyping.

Authors:  Michael T Modic
Journal:  Skeletal Radiol       Date:  2007-02       Impact factor: 2.199

4.  Phenotypic and population differences in the association between CILP and lumbar disc disease.

Authors:  I M Virtanen; Y Q Song; K M C Cheung; L Ala-Kokko; J Karppinen; D W H Ho; K D K Luk; S P Yip; J C Y Leong; K S E Cheah; P Sham; D Chan
Journal:  J Med Genet       Date:  2007-01-12       Impact factor: 6.318

Review 5.  Vertebral endplate signal changes (Modic change): a systematic literature review of prevalence and association with non-specific low back pain.

Authors:  Tue Secher Jensen; Jaro Karppinen; Joan S Sorensen; Jaakko Niinimäki; Charlotte Leboeuf-Yde
Journal:  Eur Spine J       Date:  2008-09-12       Impact factor: 3.134

6.  Decreased physical function and increased pain sensitivity in mice deficient for type IX collagen.

Authors:  Kyle D Allen; Timothy M Griffin; Ramona M Rodriguiz; William C Wetsel; Virginia B Kraus; Janet L Huebner; Lawrence M Boyd; Lori A Setton
Journal:  Arthritis Rheum       Date:  2009-09

7.  The alpha2 type IX collagen tryptophan polymorphism is associated with the severity of disc degeneration in younger patients with herniated nucleus pulposus of the lumbar spine.

Authors:  K Higashino; Y Matsui; S Yagi; Y Takata; T Goto; T Sakai; S Katoh; N Yasui
Journal:  Int Orthop       Date:  2006-04-04       Impact factor: 3.075

8.  Lumbar disc degeneration is linked to a carbohydrate sulfotransferase 3 variant.

Authors:  You-Qiang Song; Tatsuki Karasugi; Kenneth M C Cheung; Kazuhiro Chiba; Daniel W H Ho; Atsushi Miyake; Patrick Y P Kao; Kit Ling Sze; Anita Yee; Atsushi Takahashi; Yoshiharu Kawaguchi; Yasuo Mikami; Morio Matsumoto; Daisuke Togawa; Masahiro Kanayama; Dongquan Shi; Jin Dai; Qing Jiang; Chengai Wu; Wei Tian; Na Wang; John C Y Leong; Keith D K Luk; Shea-ping Yip; Stacey S Cherny; Junwen Wang; Stefan Mundlos; Anthi Kelempisioti; Pasi J Eskola; Minna Männikkö; Pirkka Mäkelä; Jaro Karppinen; Marjo-Riitta Järvelin; Paul F O'Reilly; Michiaki Kubo; Tomoatsu Kimura; Toshikazu Kubo; Yoshiaki Toyama; Hiroshi Mizuta; Kathryn S E Cheah; Tatsuhiko Tsunoda; Pak-Chung Sham; Shiro Ikegawa; Danny Chan
Journal:  J Clin Invest       Date:  2013-11       Impact factor: 14.808

9.  Structural vertebral endplate nomenclature and etiology: a study by the ISSLS Spinal Phenotype Focus Group.

Authors:  Uruj Zehra; Cora Bow; Jeffrey C Lotz; Frances M K Williams; S Rajasekaran; Jaro Karppinen; Keith D K Luk; Michele C Battiê; Dino Samartzis
Journal:  Eur Spine J       Date:  2017-09-12       Impact factor: 3.134

Review 10.  Genetic polymorphisms associated with intervertebral disc degeneration.

Authors:  Jillian E Mayer; James C Iatridis; Danny Chan; Sheeraz A Qureshi; Omri Gottesman; Andrew C Hecht
Journal:  Spine J       Date:  2013-03       Impact factor: 4.166

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