| Literature DB >> 16340011 |
Sandrine Horman1, Didier Vertommen, Richard Heath, Dietbert Neumann, Véronique Mouton, Angela Woods, Uwe Schlattner, Theo Wallimann, David Carling, Louis Hue, Mark H Rider.
Abstract
Previous studies showed that insulin antagonizes AMP-activated protein kinase activation by ischemia and that protein kinase B might be implicated. Here we investigated whether the direct phosphorylation of AMP-activated protein kinase by protein kinase B might participate in this effect. Protein kinase B phosphorylated recombinant bacterially expressed AMP-activated protein kinase heterotrimers at Ser(485) of the alpha1-subunits. In perfused rat hearts, phosphorylation of the alpha1/alpha2 AMP-activated protein kinase subunits on Ser(485)/Ser(491) was increased by insulin and insulin pretreatment decreased the phosphorylation of the alpha-subunits at Thr(172) in a subsequent ischemic episode. It is proposed that the effect of insulin to antagonize AMP-activated protein kinase activation involves a hierarchical mechanism whereby Ser(485)/Ser(491) phosphorylation by protein kinase B reduces subsequent phosphorylation of Thr(172) by LKB1 and the resulting activation of AMP-activated protein kinase.Entities:
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Year: 2005 PMID: 16340011 DOI: 10.1074/jbc.M506850200
Source DB: PubMed Journal: J Biol Chem ISSN: 0021-9258 Impact factor: 5.157