Literature DB >> 16263810

Hypercalcemia of malignancy due to ectopic transactivation of the parathyroid hormone gene.

Joshua N VanHouten1, Namyi Yu, David Rimm, Jorge Dotto, Andrew Arnold, John J Wysolmerski, Robert Udelsman.   

Abstract

CONTEXT: The physiology of PTH is well described, but regulation of PTH gene expression remains enigmatic. This is, at least in part, because of a lack of suitable cell culture systems. OBJECTIVE, DESIGN, SETTING, PATIENTS, INTERVENTIONS, AND MAIN OUTCOME MEASURES: We report a case of severe hyperparathyroidism resulting from the ectopic production of PTH by a pancreatic malignancy. Cells from the primary tumor (PEPP1 cells) were established in culture to examine the etiology of ectopic PTH gene expression in this patient. RESULTS AND
CONCLUSIONS: We failed to find amplification or rearrangement of the PTH gene but documented hypomethylation of the PTH promoter in tumor tissue. We found that PEPP1 cells support expression of a reporter gene containing regulatory sequences from the human PTH gene promoter. Therefore, this is the first report documenting ectopic PTH production by a tumor as the result of transactivation of the PTH gene. PEPP1 cells may be useful for future studies aimed at elucidating the details of PTH gene regulation.

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Year:  2005        PMID: 16263810     DOI: 10.1210/jc.2005-2095

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  12 in total

1.  Tissue-specific regulatory regions of the PTH gene localized by novel chromosome 11 rearrangement breakpoints in a parathyroid adenoma.

Authors:  Sanjay M Mallya; H Irene Wu; Elizabeth A Saria; Kristin R Corrado; Andrew Arnold
Journal:  J Bone Miner Res       Date:  2010-07-16       Impact factor: 6.741

Review 2.  Diseases of the parathyroid gland in chronic kidney disease.

Authors:  Hirotaka Komaba; Takatoshi Kakuta; Masafumi Fukagawa
Journal:  Clin Exp Nephrol       Date:  2011-08-06       Impact factor: 2.801

Review 3.  [Metabolic disorders as paraneoplastic syndromes].

Authors:  S Krug; P Michl
Journal:  Internist (Berl)       Date:  2018-02       Impact factor: 0.743

4.  Is the risk of primary hyperparathyroidism increased in patients with untreated breast cancer?

Authors:  V Belardi; E Fiore; E Giustarini; I Muller; S Sabatini; V Rosellini; E Seregni; R Agresti; C Marcocci; P Vitti; C Giani
Journal:  J Endocrinol Invest       Date:  2012-08-29       Impact factor: 4.256

5.  Ectopic secretion of parathyroid hormone in a neuroendocrine tumor: a case report and review of the literature.

Authors:  Emad Kandil; Salem Noureldine; Mohamed Abdel Khalek; Philip Daroca; Paul Friedlander
Journal:  Int J Clin Exp Med       Date:  2011-09-15

6.  [Tumor-induced hypercalcemia].

Authors:  O Hopfer; A Gawliczek; M G Kiehl
Journal:  Internist (Berl)       Date:  2013-09       Impact factor: 0.743

7.  Primary hyperparathyroidism: imaging to pathology.

Authors:  Sara Piciucchi; Domenico Barone; Giampaolo Gavelli; Alessandra Dubini; Devil Oboldi; Federica Matteuci
Journal:  J Clin Imaging Sci       Date:  2012-10-06

8.  Hyperparathyroidism with hypercalcaemia in chronic kidney disease: primary or tertiary?

Authors:  Mitchell R Lunn; Jair Muñoz Mendoza; Lezlee J Pasche; Jeffrey A Norton; Alexander L Ayco; Glenn M Chertow
Journal:  NDT Plus       Date:  2010-05-05

Review 9.  Hypercalcemia of Malignancy: An Update on Pathogenesis and Management.

Authors:  Aibek E Mirrakhimov
Journal:  N Am J Med Sci       Date:  2015-11

10.  An unusual case of malignancy-related hypercalcemia.

Authors:  Mary-Anne Doyle; Janine C Malcolm
Journal:  Int J Gen Med       Date:  2013-12-06
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