Literature DB >> 16207114

Unexplained intrauterine fetal death is accompanied by activation of complement.

Karina Richani1, Roberto Romero, Eleazar Soto, Jimmy Espinoza, Jyh Kae Nien, Tinnakorn Chaiworapongsa, Jerrie Refuerzo, Sean Blackwell, Samuel S Edwin, Joaquin Santolaya-Forgas, Moshe Mazor.   

Abstract

OBJECTIVE: Activation of the complement system has recently been implicated in the mechanisms of fetal loss in the antiphospholipid syndrome. It is, however, possible that complement activation is also involved in other causes of fetal death in the second and third trimesters of pregnancy. We therefore conducted a study to determine whether fetal death is associated with changes in the maternal plasma concentrations of complement split products or anaphylatoxins (C3a, C4a and C5a). STUDY
DESIGN: A cross-sectional study was designed to include normal pregnant women (n=60) and patients with fetal death (n=60). Patients with fetal death were classified according to the cause of fetal demise into: a) unexplained (n=44); b) associated with preeclampsia (n=8); and c) associated with chromosomal abnormalities or major congenital fetal anomalies (n=8). The plasma concentrations of C3a, C4a and C5a were measured using sensitive and specific ELISAs. Non-parametric statistics were used for analysis. A P value of <0.05 was considered significant.
RESULTS: 1) The median plasma concentration of C5a was higher in patients with fetal death than in normal pregnant women [median 16 ng/mL (range 4.5-402.5) vs. median 11.6 ng/mL (range 1.2-87.1), respectively; P<0.001]; 2) patients with an unexplained fetal death and those associated with preeclampsia had a higher median plasma C5a concentration than normal pregnant women (P=0.002 and P<0.001, respectively); 3) no differences were observed in the maternal plasma concentrations of C3a and C4a among the study groups.
CONCLUSIONS: Unexplained fetal death is associated with evidence of complement activation.

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Year:  2005        PMID: 16207114     DOI: 10.1515/JPM.2005.052

Source DB:  PubMed          Journal:  J Perinat Med        ISSN: 0300-5577            Impact factor:   1.901


  13 in total

1.  The profiles of soluble adhesion molecules in the "great obstetrical syndromes".

Authors:  Nikolina Docheva; Roberto Romero; Piya Chaemsaithong; Adi L Tarca; Gaurav Bhatti; Percy Pacora; Bogdan Panaitescu; Noppadol Chaiyasit; Tinnakorn Chaiworapongsa; Eli Maymon; Sonia S Hassan; Offer Erez
Journal:  J Matern Fetal Neonatal Med       Date:  2018-02-01

2.  Unexplained fetal death is associated with increased concentrations of anti-angiogenic factors in amniotic fluid.

Authors:  Tinnakorn Chaiworapongsa; Roberto Romero; Juan P Kusanovic; Zeynep A Savasan; Sun Kwon Kim; Shali Mazaki-Tovi; Edi Vaisbuch; Giovanna Ogge; Ichchha Madan; Zhong Dong; Lami Yeo; Pooja Mittal; Sonia S Hassan
Journal:  J Matern Fetal Neonatal Med       Date:  2010-08

3.  Unexplained fetal death has a biological signature of maternal anti-fetal rejection: chronic chorioamnionitis and alloimmune anti-human leucocyte antigen antibodies.

Authors:  JoonHo Lee; Roberto Romero; Zhong Dong; Yi Xu; Faisal Qureshi; Suzanne Jacques; Wonsuk Yoo; Tinnakorn Chaiworapongsa; Pooja Mittal; Sonia S Hassan; Chong Jai Kim
Journal:  Histopathology       Date:  2011-11       Impact factor: 5.087

4.  Activation of the alternative pathway of complement is a feature of pre-term parturition but not of spontaneous labor at term.

Authors:  Edi Vaisbuch; Roberto Romero; Offer Erez; Shali Mazaki-Tovi; Juan Pedro Kusanovic; Eleazar Soto; Zhong Dong; Tinnakorn Chaiworapongsa; Sun Kwon Kim; Giovanna Ogge; Percy Pacora; Lami Yeo; Sonia S Hassan
Journal:  Am J Reprod Immunol       Date:  2010-02-12       Impact factor: 3.886

5.  Interleukin-33 in the human placenta.

Authors:  Vanessa Topping; Roberto Romero; Nandor Gabor Than; Adi L Tarca; Zhonghui Xu; Sun Young Kim; Bing Wang; Lami Yeo; Chong Jai Kim; Sonia S Hassan; Jung-Sun Kim
Journal:  J Matern Fetal Neonatal Med       Date:  2012-11-23

6.  Preeclampsia and pregnancies with small-for-gestational age neonates have different profiles of complement split products.

Authors:  Eleazar Soto; Roberto Romero; Karina Richani; Jimmy Espinoza; Tinnakorn Chaiworapongsa; Jyh Kae Nien; Sam S Edwin; Yeon Mee Kim; Joon Seok Hong; Luis F Goncalves; Lami Yeo; Moshe Mazor; Sonia S Hassan; Juan Pedro Kusanovic
Journal:  J Matern Fetal Neonatal Med       Date:  2010-07

7.  Spontaneous abortion is associated with elevated systemic C5a and reduced mRNA of complement inhibitory proteins in placenta.

Authors:  M Banadakoppa; M S Chauhan; D Havemann; M Balakrishnan; J S Dominic; C Yallampalli
Journal:  Clin Exp Immunol       Date:  2014-09       Impact factor: 4.330

8.  Fragment Bb in amniotic fluid: evidence for complement activation by the alternative pathway in women with intra-amniotic infection/inflammation.

Authors:  Edi Vaisbuch; Roberto Romero; Offer Erez; Shali Mazaki-Tovi; Juan Pedro Kusanovic; Kusanovic Juan Pedro; Eleazar Soto; Francesca Gotsch; Zhong Dong; Tinnakorn Chaiworapongsa; Sun Kwon Kim; Pooja Mittal; Percy Pacora; Lami Yeo; Sonia S Hassan
Journal:  J Matern Fetal Neonatal Med       Date:  2009-10

9.  The C5b-9 membrane attack complex of complement activation localizes to villous trophoblast injury in vivo and modulates human trophoblast function in vitro.

Authors:  R Rampersad; A Barton; Y Sadovsky; D M Nelson
Journal:  Placenta       Date:  2008-09-09       Impact factor: 3.481

10.  Disorders of placental villous maturation in fetal death.

Authors:  Sunil Jaiman; Roberto Romero; Percy Pacora; Eunjung Jung; Gaurav Bhatti; Lami Yeo; Yeon Mee Kim; Bomi Kim; Chong Jai Kim; Jung-Sun Kim; Faisal Qureshi; Suzanne M Jacques; Offer Erez; Nardhy Gomez-Lopez; Chaur-Dong Hsu
Journal:  J Perinat Med       Date:  2020-04-01       Impact factor: 1.901

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