Literature DB >> 16197609

Safety and tolerability of PCK3145, a synthetic peptide derived from prostate secretory protein 94 (PSP94) in metastatic hormone-refractory prostate cancer.

Robert E Hawkins1, Luc Daigneault, Richard Cowan, Richard Griffiths, Chandra Panchal, Anne Armstrong, Jackie Fenemore, Alan Irvine, Kasia Sereda, Hélène Dulude.   

Abstract

BACKGROUND: The safety, tolerability, and pharmacokinetic and preliminary efficacy of PCK3145 were determined in patients with metastatic hormone-refractory prostate cancer. PATIENTS AND METHODS: PCK3145 was administered in ascending doses of 5, 20, 40, and 80 mg/m2 3 times per week for 4 weeks to cohorts of 4 patients. Dose escalation was based on dose-limiting toxicity (DLT). Pharmacokinetic profiles, tumor burden, and tumor markers (including prostate-specific antigen [PSA] and matrix metalloproteinase-9 [MMP-9] levels) were assessed. Sixteen patients received PCK3145. The median age was 66 years, and the median PSA level was 232.5 microg/L. A total of 32 cycles of therapy were administered.
RESULTS: The most common adverse events reported were pain and nausea. The only DLT was a grade 4 cardiac arrhythmia in a patient treated at the 80-mg/m2 dose level. Pharmacokinetic analysis using a 2-compartment model indicated that the mean area under the curve values increased as the dose range increased, and the mean elimination half-life ranged from 0.35 hours to 1.45 hours. The best tumor response was stable disease in 10 patients and progressive disease in 5 patients. No PSA responses were observed, but 1 patient showed a marked reduction in PSA of 41% at cycle 2. A substantial reduction in MMP-9 levels was observed in patients with baseline levels of MMP-9 > 100 microg/L.
CONCLUSION: PCK3145 was safe and well tolerated at all doses. Efficacy observations were encouraging, and the biologic activity of PCK3145 in reducing MMP-9 level may suggest a potential role of this peptide in the regulation of metastatic tumor growth.

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Year:  2005        PMID: 16197609     DOI: 10.3816/cgc.2005.n.016

Source DB:  PubMed          Journal:  Clin Prostate Cancer        ISSN: 1540-0352


  4 in total

Review 1.  Targeting tumor cell motility to prevent metastasis.

Authors:  Trenis D Palmer; William J Ashby; John D Lewis; Andries Zijlstra
Journal:  Adv Drug Deliv Rev       Date:  2011-06-02       Impact factor: 15.470

2.  A PSP94-derived peptide PCK3145 inhibits MMP-9 secretion and triggers CD44 cell surface shedding: implication in tumor metastasis.

Authors:  Borhane Annabi; Mounia Bouzeghrane; Jean-Christophe Currie; Robert Hawkins; Hélène Dulude; Luc Daigneault; Marcia Ruiz; Jan Wisniewski; Seema Garde; Shafaat A Rabbani; Chandra Panchal; Jinzi J Wu; Richard Béliveau
Journal:  Clin Exp Metastasis       Date:  2005       Impact factor: 5.150

Review 3.  Drug discovery in advanced prostate cancer: translating biology into therapy.

Authors:  Timothy A Yap; Alan D Smith; Roberta Ferraldeschi; Bissan Al-Lazikani; Paul Workman; Johann S de Bono
Journal:  Nat Rev Drug Discov       Date:  2016-07-22       Impact factor: 84.694

4.  Loss of PSP94 expression is associated with early PSA recurrence and deteriorates outcome of PTEN deleted prostate cancers.

Authors:  Andreas M Luebke; Ali Attarchi-Tehrani; Jan Meiners; Claudia Hube-Magg; Dagmar S Lang; Martina Kluth; Maria Christina Tsourlakis; Sarah Minner; Ronald Simon; Guido Sauter; Franziska Büscheck; Frank Jacobsen; Andrea Hinsch; Stefan Steurer; Thorsten Schlomm; Hartwig Huland; Markus Graefen; Alexander Haese; Hans Heinzer; Till S Clauditz; Eike Burandt; Waldemar Wilczak; Doris Höflmayer
Journal:  Cancer Biol Med       Date:  2019-05       Impact factor: 4.248

  4 in total

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