Literature DB >> 16169479

Signal transduction: how rad53 kinase is activated.

Achille Pellicioli1, Marco Foiani.   

Abstract

Recent studies have elucidated the activation mechanism of the Rad53 checkpoint kinase and the role of Rad9-like adaptor proteins in mediating signal transduction from PIKK sensor kinases that detect DNA damage to the effector kinases that play a part in mending that damage.

Entities:  

Mesh：

Substances：

Year:  2005        PMID： 16169479     DOI： 10.1016/j.cub.2005.08.057

Source DB:  PubMed          Journal:  Curr Biol        ISSN： 0960-9822            Impact factor:   10.834


  43 in total

1.  Saccharomyces cerevisiae Dbf4 has unique fold necessary for interaction with Rad53 kinase.

Authors:  Lindsay A Matthews; Darryl R Jones; Ajai A Prasad; Bernard P Duncker; Alba Guarné
Journal:  J Biol Chem       Date:  2011-11-29       Impact factor: 5.157

2.  Mechanisms of checkpoint kinase Rad53 inactivation after a double-strand break in Saccharomyces cerevisiae.

Authors:  Ghislaine Guillemain; Emilie Ma; Sarah Mauger; Simona Miron; Robert Thai; Raphaël Guérois; Françoise Ochsenbein; Marie-Claude Marsolier-Kergoat
Journal:  Mol Cell Biol       Date:  2007-02-26       Impact factor: 4.272

3.  Activation of protein kinase Tel1 through recognition of protein-bound DNA ends.

Authors:  Kenzo Fukunaga; Youngho Kwon; Patrick Sung; Katsunori Sugimoto
Journal:  Mol Cell Biol       Date:  2011-03-14       Impact factor: 4.272

4.  Dampening DNA damage checkpoint signalling via coordinated BRCT domain interactions.

Authors:  José R Cussiol; Carolyn M Jablonowski; Askar Yimit; Grant W Brown; Marcus B Smolka
Journal:  EMBO J       Date:  2015-04-20       Impact factor: 11.598

Review 5.  Dbf4: the whole is greater than the sum of its parts.

Authors:  Lindsay A Matthews; Alba Guarné
Journal:  Cell Cycle       Date:  2013-04-02       Impact factor: 4.534

Review 6.  Coupling end resection with the checkpoint response at DNA double-strand breaks.

Authors:  Matteo Villa; Corinne Cassani; Elisa Gobbini; Diego Bonetti; Maria Pia Longhese
Journal:  Cell Mol Life Sci       Date:  2016-05-03       Impact factor: 9.261

7.  FACT prevents the accumulation of free histones evicted from transcribed chromatin and a subsequent cell cycle delay in G1.

Authors:  Macarena Morillo-Huesca; Douglas Maya; Mari Cruz Muñoz-Centeno; Rakesh Kumar Singh; Vincent Oreal; Gajjalaiahvari Ugander Reddy; Dun Liang; Vincent Géli; Akash Gunjan; Sebastián Chávez
Journal:  PLoS Genet       Date:  2010-05-20       Impact factor: 5.917

8.  Dynamics of Rad9 chromatin binding and checkpoint function are mediated by its dimerization and are cell cycle-regulated by CDK1 activity.

Authors:  Magda Granata; Federico Lazzaro; Daniele Novarina; Davide Panigada; Fabio Puddu; Carla Manuela Abreu; Ramesh Kumar; Muriel Grenon; Noel F Lowndes; Paolo Plevani; Marco Muzi-Falconi
Journal:  PLoS Genet       Date:  2010-08-05       Impact factor: 5.917

9.  Rad17 plays a central role in establishment of the interaction between TopBP1 and the Rad9-Hus1-Rad1 complex at stalled replication forks.

Authors:  Joon Lee; William G Dunphy
Journal:  Mol Biol Cell       Date:  2010-01-28       Impact factor: 4.138

10.  Elevated levels of the polo kinase Cdc5 override the Mec1/ATR checkpoint in budding yeast by acting at different steps of the signaling pathway.

Authors:  Roberto Antonio Donnianni; Matteo Ferrari; Federico Lazzaro; Michela Clerici; Benjamin Tamilselvan Nachimuthu; Paolo Plevani; Marco Muzi-Falconi; Achille Pellicioli
Journal:  PLoS Genet       Date:  2010-01-22       Impact factor: 5.917
View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.