Literature DB >> 16129795

Subunit interaction determines IKs participation in cardiac repolarization and repolarization reserve.

Jonathan Silva1, Yoram Rudy.   

Abstract

BACKGROUND: The role of IKs, the slow delayed rectifier K+ current, in cardiac ventricular repolarization has been a subject of debate. METHODS AND
RESULTS: We develop a detailed Markov model of IKs and its alpha-subunit KCNQ1 and examine their kinetic properties during the cardiac ventricular action potential at different rates. We observe that interaction between KCNQ1 and KCNE1 (the beta-subunit) confers kinetic properties on IKs that make it suitable for participation in action potential repolarization and its adaptation to rate changes; in particular, the channel develops an available reserve of closed states near the open state that can open rapidly on demand.
CONCLUSIONS: Because of its ability to form an available reserve, IKs can function as a repolarization reserve when IKr, the rapid delayed rectifier, is reduced by disease or drug and can prevent excessive action potential prolongation and development of arrhythmogenic early afterdepolarizations.

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Year:  2005        PMID: 16129795      PMCID: PMC1820744          DOI: 10.1161/CIRCULATIONAHA.105.543306

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  21 in total

1.  Effects of IKr and IKs heterogeneity on action potential duration and its rate dependence: a simulation study.

Authors:  P C Viswanathan; R M Shaw; Y Rudy
Journal:  Circulation       Date:  1999-05-11       Impact factor: 29.690

2.  Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel.

Authors:  M C Sanguinetti; M E Curran; A Zou; J Shen; P S Spector; D L Atkinson; M T Keating
Journal:  Nature       Date:  1996-11-07       Impact factor: 49.962

3.  Early afterdepolarizations in cardiac myocytes: mechanism and rate dependence.

Authors:  J Zeng; Y Rudy
Journal:  Biophys J       Date:  1995-03       Impact factor: 4.033

4.  Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits.

Authors:  M Tristani-Firouzi; M C Sanguinetti
Journal:  J Physiol       Date:  1998-07-01       Impact factor: 5.182

5.  A simple model of K+ channel activation in nerve membrane.

Authors:  J R Clay
Journal:  J Theor Biol       Date:  1995-07-12       Impact factor: 2.691

6.  Evidence for the presence of M cells in the guinea pig ventricle.

Authors:  S Sicouri; M Quist; C Antzelevitch
Journal:  J Cardiovasc Electrophysiol       Date:  1996-06

7.  Mutations in the hminK gene cause long QT syndrome and suppress IKs function.

Authors:  I Splawski; M Tristani-Firouzi; M H Lehmann; M C Sanguinetti; M T Keating
Journal:  Nat Genet       Date:  1997-11       Impact factor: 38.330

8.  Shaker potassium channel gating. III: Evaluation of kinetic models for activation.

Authors:  W N Zagotta; T Hoshi; R W Aldrich
Journal:  J Gen Physiol       Date:  1994-02       Impact factor: 4.086

9.  Gating of IsK expressed in Xenopus oocytes depends on the amount of mRNA injected.

Authors:  J Cui; R P Kline; P Pennefather; I S Cohen
Journal:  J Gen Physiol       Date:  1994-07       Impact factor: 4.086

10.  Shaker potassium channel gating. II: Transitions in the activation pathway.

Authors:  W N Zagotta; T Hoshi; J Dittman; R W Aldrich
Journal:  J Gen Physiol       Date:  1994-02       Impact factor: 4.086

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  93 in total

1.  Role of the transient outward potassium current in the genesis of early afterdepolarizations in cardiac cells.

Authors:  Zhenghang Zhao; Yuanfang Xie; Hairuo Wen; Dandan Xiao; Charelle Allen; Nadezhda Fefelova; Wen Dun; Penelope A Boyden; Zhilin Qu; Lai-Hua Xie
Journal:  Cardiovasc Res       Date:  2012-06-01       Impact factor: 10.787

2.  Early afterdepolarizations and cardiac arrhythmias.

Authors:  James N Weiss; Alan Garfinkel; Hrayr S Karagueuzian; Peng-Sheng Chen; Zhilin Qu
Journal:  Heart Rhythm       Date:  2010-09-22       Impact factor: 6.343

3.  KCNQ1 channels voltage dependence through a voltage-dependent binding of the S4-S5 linker to the pore domain.

Authors:  Frank S Choveau; Nicolas Rodriguez; Fayal Abderemane Ali; Alain J Labro; Thierry Rose; Shehrazade Dahimène; Hélène Boudin; Carole Le Hénaff; Denis Escande; Dirk J Snyders; Flavien Charpentier; Jean Mérot; Isabelle Baró; Gildas Loussouarn
Journal:  J Biol Chem       Date:  2010-10-12       Impact factor: 5.157

4.  Arrhythmia formation in subclinical ("silent") long QT syndrome requires multiple insults: quantitative mechanistic study using the KCNQ1 mutation Q357R as example.

Authors:  Thomas O'Hara; Yoram Rudy
Journal:  Heart Rhythm       Date:  2011-09-25       Impact factor: 6.343

Review 5.  Drug-induced long QT syndrome.

Authors:  Prince Kannankeril; Dan M Roden; Dawood Darbar
Journal:  Pharmacol Rev       Date:  2010-12       Impact factor: 25.468

Review 6.  Voltage-Dependent Gating: Novel Insights from KCNQ1 Channels.

Authors:  Jianmin Cui
Journal:  Biophys J       Date:  2016-01-05       Impact factor: 4.033

7.  Modelling and imaging cardiac repolarization abnormalities.

Authors:  Y Rudy
Journal:  J Intern Med       Date:  2006-01       Impact factor: 8.989

Review 8.  Computational biology in the study of cardiac ion channels and cell electrophysiology.

Authors:  Yoram Rudy; Jonathan R Silva
Journal:  Q Rev Biophys       Date:  2006-07-19       Impact factor: 5.318

9.  Voltage-dependent activation in EAG channels follows a ligand-receptor rather than a mechanical-lever mechanism.

Authors:  Olfat A Malak; Grigory S Gluhov; Anastasia V Grizel; Kseniya S Kudryashova; Olga S Sokolova; Gildas Loussouarn
Journal:  J Biol Chem       Date:  2019-02-26       Impact factor: 5.157

Review 10.  Impact of ancillary subunits on ventricular repolarization.

Authors:  Geoffrey W Abbott; Xianghua Xu; Torsten K Roepke
Journal:  J Electrocardiol       Date:  2007 Nov-Dec       Impact factor: 1.438

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