Literature DB >> 16121967

Clazosentan (AXV-034343), a selective endothelin A receptor antagonist, in the prevention of cerebral vasospasm following severe aneurysmal subarachnoid hemorrhage: results of a randomized, double-blind, placebo-controlled, multicenter phase IIa study.

Peter Vajkoczy1, Bernhard Meyer, Stefan Weidauer, Andreas Raabe, Claudius Thome, Florian Ringel, Volker Breu, Peter Schmiedek.   

Abstract

OBJECTIVE: The goal of this study was to investigate the safety and tolerability of the novel endothelin A (ETA) receptor antagonist clazosentan in patients with subarachnoid hemorrhage (SAH) and its potential to reduce the incidence and severity of cerebral vasospasm following surgical clipping of the aneurysm.
METHODS: This Phase IIa multicenter study had two parts: a double-blind, randomized Part A (some patients given clazosentan [0.2 mg/kg/hr] and others given placebo), in which statistical inference was performed, and an open-label Part B (patients with established vasospasm given clazosentan [0.4 mg/kg/hr for 12 hours followed by 0.2 mg/kg/hr]) for exploratory purposes only. Primary end points were the incidence and severity of angiographic vasospasm on Day 8 after SAH and the safety and tolerability of the drug. Thirty-four patients (Hunt and Hess Grades III and IV and Fisher Grade > or = 3) were recruited and 32 (15 in the clazosentan group and 17 in the placebo group) were retained in the intent-to-treat population; 19 patients entered Part B. In Part A, treatment with clazosentan resulted in a reduced incidence of angiographically evident cerebral vasospasm (40% compared with 88% of patients, p = 0.008). In addition, the severity of vasospasm was reduced in the clazosentan group (p = 0.012). In Part B of the study, in 50% of assessable patients who were initially treated with placebo reversal of vasospasm was observed following the initiation of clazosentan therapy. The incidence of new infarctions was 15% in the clazosentan group and 44% in the placebo group (p = 0.130). There was no adverse event pattern indicating a specific organ toxicity of clazosentan.
CONCLUSIONS: This study indicates that clazosentan reduces the frequency and severity of cerebral vasospasm following severe aneurysmal SAH with the incidence and severity of adverse events comparable to that of placebo.

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Year:  2005        PMID: 16121967     DOI: 10.3171/jns.2005.103.1.0009

Source DB:  PubMed          Journal:  J Neurosurg        ISSN: 0022-3085            Impact factor:   5.115


  70 in total

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2.  Tolerability, pharmacokinetics, and pharmacodynamics of clazosentan, a parenteral endothelin receptor antagonist.

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4.  Recombinant osteopontin in cerebral vasospasm after subarachnoid hemorrhage.

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5.  Role of endothelin-1 in human aneurysmal subarachnoid hemorrhage: associations with vasospasm and delayed cerebral ischemia.

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Review 7.  [Aneurysmal subarachnoid hemorrhage. Significance and complications].

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8.  Cerebral inflammatory response and predictors of admission clinical grade after aneurysmal subarachnoid hemorrhage.

Authors:  Khalid A Hanafy; R Morgan Stuart; Luis Fernandez; J Michael Schmidt; Jan Claassen; Kiwon Lee; E Sander Connolly; Stephan A Mayer; Neeraj Badjatia
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Review 9.  Cerebral vasospasm following subarachnoid hemorrhage: time for a new world of thought.

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Journal:  Neurol Res       Date:  2009-03       Impact factor: 2.448

10.  Role of gap junctions in early brain injury following subarachnoid hemorrhage.

Authors:  Robert Ayer; Wanqiu Chen; Takashi Sugawara; Hidenori Suzuki; John H Zhang
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