Literature DB >> 16105963

High frequency of skewed X-chromosome inactivation in females with autoimmune thyroid disease: a possible explanation for the female predisposition to thyroid autoimmunity.

Thomas Heiberg Brix1, Gun Peggy S Knudsen, Marianne Kristiansen, Kirsten Ohm Kyvik, Karen Helene Orstavik, Laszlo Hegedüs.   

Abstract

CONTEXT: Autoimmune thyroid diseases (AITD) comprise Graves' disease (GD) and Hashimoto's thyroiditis (HT). They are characterized by loss of immunological self-tolerance and female preponderance. Theoretically, X chromosome inactivation (XCI) and resultant tissue chimerism could offer an explanation for the female predisposition to AITD. AIM: Our aim was to examine whether skewed XCI is associated with AITD. DESIGNS: We first conducted a classical case-control study of twin individuals with and without AITD, and then a case-control study of twin pairs discordant for AITD. PARTICIPANTS: Participants included 32 female twins with AITD and a control group of 96 healthy female twin individuals.
METHODS: XCI analysis was performed by enzymatic predigestion of DNA with a methylation-sensitive enzyme followed by PCR of the polymorphic CAG repeat of the androgen receptor gene. The XCI pattern was classified as skewed when 80% or more of the cells preferentially inactivated the same X chromosome. MAIN OUTCOME MEASURES: We assessed the prevalence of skewed XCI.
RESULTS: The frequency of skewed XCI in female twins with AITD, GD, and HT was 34, 37, and 31%, respectively, which was higher than the prevalence in the corresponding control populations, 11% (P = 0.003), 14% (P = 0.045), and 8% (P = 0.057), respectively. Similar results were found in twin pairs discordant for AITD. Overall, skewed XCI was associated with an increased risk of developing AITD, with an odds ratio of 9.0 (95% confidence interval, 1.64-49.4) (P = 0.022).
CONCLUSION: These observations suggest a possible role of XCI in the etiology of AITD and may in part explain the female preponderance of AITD.

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Year:  2005        PMID: 16105963     DOI: 10.1210/jc.2005-1366

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  49 in total

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Authors:  Elif Uz; Ismail Dolen; Atakan R Al; Tayfun Ozcelik
Journal:  Hum Genet       Date:  2006-11-07       Impact factor: 4.132

Review 2.  X-linked clonality testing: interpretation and limitations.

Authors:  George L Chen; Josef T Prchal
Journal:  Blood       Date:  2007-04-13       Impact factor: 22.113

Review 3.  X chromosome inactivation and female predisposition to autoimmunity.

Authors:  Tayfun Ozcelik
Journal:  Clin Rev Allergy Immunol       Date:  2008-06       Impact factor: 8.667

4.  The relationship between skewed X chromosome inactivation and the prognosis of Graves' and Hashimoto's diseases.

Authors:  Naoko Ishido; Naoya Inoue; Mikio Watanabe; Yoh Hidaka; Yoshinori Iwatani
Journal:  Thyroid       Date:  2014-12-10       Impact factor: 6.568

5.  Genes known to escape X chromosome inactivation predict co-morbid chronic musculoskeletal pain and posttraumatic stress symptom development in women following trauma exposure.

Authors:  Shan Yu; Constance Chen; Yue Pan; Michael C Kurz; Elizabeth Datner; Phyllis L Hendry; Marc-Anthony Velilla; Christopher Lewandowski; Claire Pearson; Robert Domeier; Samuel A McLean; Sarah D Linnstaedt
Journal:  Am J Med Genet B Neuropsychiatr Genet       Date:  2018-12-11       Impact factor: 3.568

Review 6.  Sex-related factors in autoimmune liver diseases.

Authors:  Dorothee Schwinge; Christoph Schramm
Journal:  Semin Immunopathol       Date:  2018-10-01       Impact factor: 9.623

Review 7.  Sexual dimorphism in autoimmunity.

Authors:  Kira Rubtsova; Philippa Marrack; Anatoly V Rubtsov
Journal:  J Clin Invest       Date:  2015-04-27       Impact factor: 14.808

8.  Preliminary evidence of a noncausal association between the X-chromosome inactivation pattern and thyroid autoimmunity: a twin study.

Authors:  Thomas Heiberg Brix; Pia Skov Hansen; Kirsten Ohm Kyvik; Laszlo Hegedüs
Journal:  Eur J Hum Genet       Date:  2009-09-30       Impact factor: 4.246

9.  Expanded clinical phenotype of women with the FMR1 premutation.

Authors:  Sarah M Coffey; Kylee Cook; Nicole Tartaglia; Flora Tassone; Danh V Nguyen; Ruiqin Pan; Hannah E Bronsky; Jennifer Yuhas; Mariya Borodyanskaya; Jim Grigsby; Melanie Doerflinger; Paul J Hagerman; Randi J Hagerman
Journal:  Am J Med Genet A       Date:  2008-04-15       Impact factor: 2.802

10.  Analysis of skewed X-chromosome inactivation in females with rheumatoid arthritis and autoimmune thyroid diseases.

Authors:  Ghazi Chabchoub; Elif Uz; Abdellatif Maalej; Chigdem A Mustafa; Ahmed Rebai; Mouna Mnif; Zouheir Bahloul; Nadir R Farid; Tayfun Ozcelik; Hammadi Ayadi
Journal:  Arthritis Res Ther       Date:  2009-07-09       Impact factor: 5.156

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