Literature DB >> 16098430

Endothelial vasomotor dysfunction in the brachial artery is associated with late in-stent coronary restenosis.

Yoshinobu Kitta1, Takamitsu Nakamura, Yasushi Kodama, Hajime Takano, Ken Umetani, Daisuke Fujioka, Yukio Saito, Ken-ichi Kawabata, Jyun-ei Obata, Yoshihide Ichigi, Akira Mende, Kiyotaka Kugiyama.   

Abstract

OBJECTIVES: This study examined whether endothelial dysfunction in the brachial artery might be associated with late in-stent restenosis (ISR) after percutaneous coronary intervention (PCI).
BACKGROUND: Simple and noninvasive identification of late ISR might help to select patients who require further angiographic evaluation.
METHODS: Endothelium-dependent flow-mediated dilation (FMD) of the brachial artery was measured before (initial FMD) and at six months (follow-up FMD) after PCI in 141 consecutive patients who had elective and successful PCI with bare metal stents in de novo lesions of native coronary arteries for symptomatic coronary artery disease. Follow-up angiography was performed at six months after PCI in all patients.
RESULTS: With multivariate logistic regression analysis, the impairment (< or = 4.8% dilation from baseline diameter) of FMD at follow-up showed the strongest association with late ISR (defined as > 50% diameter stenosis, n = 46) independently of other clinical and angiographic variables known to be associated with ISR (odds ratio 7.4, 95% confidence interval 2.8 to 19.2, p < 0.001), whereas the initial FMD did not have the association. The sensitivity of impaired FMD at follow-up (69%) in detecting ISR was higher than chest pain during the follow-up period (45%), with comparable specificity. The impaired FMD in combination with the chest pain increased the sensitivity to 90%.
CONCLUSIONS: The impairment of FMD in the brachial artery at the time of follow-up was independently and closely associated with late ISR in native coronary arteries. The noninvasive assessment of FMD at the time of follow-up might be useful for identification of late ISR.

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Year:  2005        PMID: 16098430     DOI: 10.1016/j.jacc.2005.04.055

Source DB:  PubMed          Journal:  J Am Coll Cardiol        ISSN: 0735-1097            Impact factor:   24.094


  15 in total

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