Literature DB >> 16025877

Regional body fat distribution in HIV-infected patients with lipodystrophy.

Warren L Dinges1, Dali Chen, Peter G Snell, Paul T Weatherall, Dolores M Peterson, Abhimanyu Garg.   

Abstract

BACKGROUND: Objective criteria for the assessment of patients with lipodystrophy syndrome in human immunodeficiency virus infection (LDHIV) have not emerged.
METHODS: We compared regional body fat changes in 13 men with severe LDHIV on protease inhibitor-inclusive antiretroviral therapy with 13 control HIV-infected men using anthropometry, dual-energy X-ray absorptiometry (DEXA), and whole-body magnetic resonance imaging (MRI).
RESULTS: LDHIV patients, compared with control subjects, had thinner gluteal, suprailiac, and triceps skinfolds (p < .01) and increased waist circumference (98 +/- 5 cm vs 86 +/- 9 cm, respectively; p = .0008). DEXA studies revealed reduced lower extremity fat (12 +/- 5% vs 22 +/- 9%; p = .0006), increased head and neck fat (18 +/- 3% vs 16 +/- 1%; p = .01), and increased proportion of total body fat in the trunk (65 +/- 7% vs 53 +/- 8%; p = .0005). MRI analysis revealed reduced thigh fat (12 +/- 5% vs 22 +/- 12%; p = .01), increased dorsocervical fat depth (47 +/- 24 mm vs 19 +/- 7 mm; p = .0009), and nearly significant increase in intra-abdominal fat (218 +/- 90 cm2 vs 157 +/- 70 cm2; p = .057). Interestingly, control subjects showed a positive relationship between intra-abdominal and dorsocervical fat (r= .57, p = .04), but the LDHIV patients showed a negative relationship (r= -.55, p = .05), suggesting a novel split phenotype among LDHIV patients of either dorsocervical or intra-abdominal fat accumulation.
CONCLUSIONS: We conclude that MRI provides the best tools for definition of LDHIV syndrome and reveals variable phenotypes among LDHIV patients.

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Year:  2005        PMID: 16025877     DOI: 10.2310/6650.2005.00001

Source DB:  PubMed          Journal:  J Investig Med        ISSN: 1081-5589            Impact factor:   2.895


  6 in total

1.  Assessment of body fat composition disturbances by bioimpedance analysis in HIV-infected adults.

Authors:  P Freitas; D Carvalho; A C Santos; J Mesquita; F Correia; S Xerinda; R Marques; E Martinez; A Sarmento; J L Medina
Journal:  J Endocrinol Invest       Date:  2011-07-05       Impact factor: 4.256

Review 2.  Obesity, insulin resistance and comorbidities? Mechanisms of association.

Authors:  Ana Valeria B Castro; Cathryn M Kolka; Stella P Kim; Richard N Bergman
Journal:  Arq Bras Endocrinol Metabol       Date:  2014-08

3.  Growth hormone and tesamorelin in the management of HIV-associated lipodystrophy.

Authors:  Roger Bedimo
Journal:  HIV AIDS (Auckl)       Date:  2011-07-10

4.  Predictors of acquired lipodystrophy in juvenile-onset dermatomyositis and a gradient of severity.

Authors:  April Bingham; Gulnara Mamyrova; Kristina I Rother; Elif Oral; Elaine Cochran; Ahalya Premkumar; David Kleiner; Laura James-Newton; Ira N Targoff; Janardan P Pandey; Danielle Mercatante Carrick; Nancy Sebring; Terrance P O'Hanlon; Maria Ruiz-Hidalgo; Maria Turner; Leslie B Gordon; Jorge Laborda; Steven R Bauer; Perry J Blackshear; Lisa Imundo; Frederick W Miller; Lisa G Rider
Journal:  Medicine (Baltimore)       Date:  2008-03       Impact factor: 1.817

5.  Study of Body Composition and Metabolic Parameters in HIV-1 Male Patients.

Authors:  Gurudath Gundurao Sreekantamurthy; N Biplab Singh; Th Bhimo Singh; Th Suraj Singh; Karam Romeo Singh
Journal:  J Nutr Metab       Date:  2014-06-10

6.  Endoplasmic reticulum stress activation in adipose tissue induces metabolic syndrome in individuals with familial partial lipodystrophy of the Dunnigan type.

Authors:  Maria C Foss-Freitas; Rafael C Ferraz; Luciana Z Monteiro; Patricia M Gomes; Ricardo Iwakura; Luiz Carlos C de Freitas; Milton C Foss
Journal:  Diabetol Metab Syndr       Date:  2018-02-09       Impact factor: 3.320

  6 in total

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