Literature DB >> 15976787

Periodontal treatment improves endothelial dysfunction in patients with severe periodontitis.

Gerald Seinost1, Gernot Wimmer, Martina Skerget, Erik Thaller, Marianne Brodmann, Robert Gasser, Rudolf O Bratschko, Ernst Pilger.   

Abstract

BACKGROUND: Because epidemiological studies provide evidence that periodontal infections are associated with an increased risk of progression of cardiovascular and cerebrovascular disease, we postulated that endothelial dysfunction, a critical element in the pathogenesis of atherosclerosis, would be present in patients with periodontal disease.
METHODS: We tested endothelial function in 30 patients with severe periodontitis and 31 control subjects using flow-mediated dilation (FMD) of the brachial artery. The groups were matched for age, sex, and cardiovascular risk factors. Three months after periodontal treatment, including both mechanical and pharmacological therapy, endothelial function was reassessed by brachial artery FMD. Markers of systemic inflammation were measured at baseline and at follow up.
RESULTS: Flow-mediated dilation was significantly lower in patients with periodontitis than in control subjects (6.1% +/- 4.4% vs 8.5% +/- 3.4%, P = .002). Successful periodontal treatment resulted in a significant improvement in FMD (9.8% +/- 5.7%; P = .003 compared to baseline) accompanied by a significant decrease in C-reactive protein concentrations (1.1 +/- 1.9 vs 0.8 +/- 0.8 at baseline, P = .026). Endothelium-independent nitro-induced vasodilation did not differ between the study groups at baseline or after periodontal therapy.
CONCLUSION: These results indicate that treatment of severe periodontitis reverses endothelial dysfunction. Whether improved endothelial function will translate into a beneficial effect on atherogenesis and cardiovascular events needs further investigation.

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Year:  2005        PMID: 15976787     DOI: 10.1016/j.ahj.2004.09.059

Source DB:  PubMed          Journal:  Am Heart J        ISSN: 0002-8703            Impact factor:   4.749


  52 in total

1.  Periodontal therapy alters gene expression of peripheral blood monocytes.

Authors:  Panos N Papapanou; Michael H Sedaghatfar; Ryan T Demmer; Dana L Wolf; Jun Yang; Georg A Roth; Romanita Celenti; Paul B Belusko; Evanthia Lalla; Paul Pavlidis
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Review 3.  Relationship between vascular endothelium and periodontal disease in atherosclerotic lesions: Review article.

Authors:  Marco Aurélio Lumertz Saffi; Mariana Vargas Furtado; Carisi Anne Polanczyk; Márlon Munhoz Montenegro; Ingrid Webb Josephson Ribeiro; Cassio Kampits; Alex Nogueira Haas; Cassiano Kuchenbecker Rösing; Eneida Rejane Rabelo-Silva
Journal:  World J Cardiol       Date:  2015-01-26

4.  Arterial stiffness in periodontitis patients and controls. A case–control and pilot intervention study.

Authors:  W Houcken; W J Teeuw; S Bizzarro; E Alvarez Rodriguez; T A Mulders; B-Jh van den Born; B G Loos
Journal:  J Hum Hypertens       Date:  2015-05-14       Impact factor: 3.012

Review 5.  Inflammatory mechanisms linking periodontal diseases to cardiovascular diseases.

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Journal:  J Clin Periodontol       Date:  2013-04       Impact factor: 8.728

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Journal:  J Biol Chem       Date:  2011-05-12       Impact factor: 5.157

7.  Evaluation of serum anti-cardiolipin antibodies after non-surgical periodontal treatment in chronic periodontitis patients.

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Journal:  Odontology       Date:  2014-02-14       Impact factor: 2.634

8.  Results from the Periodontitis and Vascular Events (PAVE) Study: a pilot multicentered, randomized, controlled trial to study effects of periodontal therapy in a secondary prevention model of cardiovascular disease.

Authors:  Steven Offenbacher; James D Beck; Kevin Moss; Luisito Mendoza; David W Paquette; David A Barrow; David J Couper; Dawn D Stewart; Karen L Falkner; Susan P Graham; Sara Grossi; John C Gunsolley; Theresa Madden; Gerardo Maupome; Maurizio Trevisan; Thomas E Van Dyke; Robert J Genco
Journal:  J Periodontol       Date:  2009-02       Impact factor: 6.993

9.  Porphyromonas gingivalis infection and prothrombotic effects in human aortic smooth muscle cells.

Authors:  Georg A Roth; Klaus Aumayr; Mary Beth Giacona; Panos N Papapanou; Ann Marie Schmidt; Evanthia Lalla
Journal:  Thromb Res       Date:  2008-09-11       Impact factor: 3.944

10.  Treponema denticola alters cell vitality and induces HO-1 and Hsp70 expression in porcine aortic endothelial cells.

Authors:  Chiara Bernardini; Paolo Gaibani; Augusta Zannoni; Caterina Vocale; Maria Laura Bacci; Gabriela Piana; Monica Forni; Vittorio Sambri
Journal:  Cell Stress Chaperones       Date:  2009-12-20       Impact factor: 3.667

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