Literature DB >> 15954088

Critical window of male reproductive tract development in rats following gestational exposure to di-n-butyl phthalate.

Christina M Carruthers1, Paul M D Foster.   

Abstract

BACKGROUND: Gestational exposure to di-n-butyl phthalate (DBP), a ubiquitous environmental contaminant, has been shown to interfere with the development of the male reproductive tract by acting as an antiandrogen. This study was conducted to identify the critical days for the abnormal development of the male reproductive tract, specifically the testis and epididymis.
METHODS: Timed-pregnant Sprague-Dawley rats were dosed with DBP at 500 mg/kg/day on gestation day (GD) 14 and 15, 15 and 16, 16 and 17, 17 and 18, 18 and 19, or 19 and 20 (GD 0=plug day). Anogenital distance (AGD) was measured on postnatal day (PND) 1 and 13, while areloa number was recorded on PND 13 only. After weaning, males were allowed to mature to PND 90 at which time they were necropsied. Areloa number and AGD were recorded and testes, epididymides, seminal vesicles, prostate gland, kidneys, and liver weighed. Blood serum was collected and assayed for total testosterone concentration.
RESULTS: There were no observable effects on litter size, sex ratio, serum testosterone concentration, or mortality of pups. Statistically significant permanent reductions in AGD were seen in males exposed prenatally to DBP on GD 15 and 16 or GD 18 and 19. On PND 13, areola were present in males exposed to DBP on GD 15 and 16, 16 and 17, 17 and 18, and 19 and 20. However, permanent retention occurred only in males after DBP exposure on GD 16 and 17. Exposure to DBP on only GD 17 and 18 elicited a reduction in epididymal weights; while exposure on only GD 16 and 17 caused a significant increase in the weights of the testes due to edema. In this study, epididymal and testicular malformations were most prevalent after exposure to DBP on any gestational day. Epididymal malformations, characterized by agenesis of various regions and small or flaccid testes were significantly increased in DBP-exposed males only on GD 16 and 17.
CONCLUSIONS: These findings suggest that 2-day DBP exposure is highly detrimental to the developing reproductive tract of the male fetus and the critical window for abnormal development is GD 16-18.

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Year:  2005        PMID: 15954088     DOI: 10.1002/bdrb.20050

Source DB:  PubMed          Journal:  Birth Defects Res B Dev Reprod Toxicol        ISSN: 1542-9733


  35 in total

1.  Effects of prenatal exposure to a low dose atrazine metabolite mixture on pubertal timing and prostate development of male Long-Evans rats.

Authors:  Jason P Stanko; Rolondo R Enoch; Jennifer L Rayner; Christine C Davis; Douglas C Wolf; David E Malarkey; Suzanne E Fenton
Journal:  Reprod Toxicol       Date:  2010-08-19       Impact factor: 3.143

Review 2.  Cumulative effects of antiandrogenic chemical mixtures and their relevance to human health risk assessment.

Authors:  Kembra L Howdeshell; Andrew K Hotchkiss; L Earl Gray
Journal:  Int J Hyg Environ Health       Date:  2016-11-19       Impact factor: 5.840

3.  In utero exposure to simvastatin reduces postnatal survival and permanently alters reproductive tract development in the Crl:CD(SD) male rat.

Authors:  Brandiese E J Beverly; Johnathan R Furr; Christy S Lambright; Vickie S Wilson; Barry S McIntyre; Paul M D Foster; Greg Travlos; L Earl Gray
Journal:  Toxicol Appl Pharmacol       Date:  2019-01-11       Impact factor: 4.219

Review 4.  Interpretation of studies on the developmental reproductive toxicology of 2,3,7,8-tetrachlorodibenzo-p-dioxin in male offspring.

Authors:  David R Bell; Sally Clode; Ming Qi Fan; Alwyn Fernandes; Paul M D Foster; Tao Jiang; George Loizou; Alan MacNicoll; Brian G Miller; Martin Rose; Lang Tran; Shaun White
Journal:  Food Chem Toxicol       Date:  2010-04-11       Impact factor: 6.023

5.  Genomic biomarkers of phthalate-induced male reproductive developmental toxicity: a targeted RT-PCR array approach for defining relative potency.

Authors:  Bethany R Hannas; Christy S Lambright; Johnathan Furr; Nicola Evans; Paul M D Foster; Earl L Gray; Vickie S Wilson
Journal:  Toxicol Sci       Date:  2011-11-22       Impact factor: 4.849

Review 6.  Of mice and men (and rats): phthalate-induced fetal testis endocrine disruption is species-dependent.

Authors:  Kamin J Johnson; Nicholas E Heger; Kim Boekelheide
Journal:  Toxicol Sci       Date:  2012-06-14       Impact factor: 4.849

7.  Dipentyl phthalate dosing during sexual differentiation disrupts fetal testis function and postnatal development of the male Sprague-Dawley rat with greater relative potency than other phthalates.

Authors:  Bethany R Hannas; Johnathan Furr; Christy S Lambright; Vickie S Wilson; Paul M D Foster; L Earl Gray
Journal:  Toxicol Sci       Date:  2010-12-20       Impact factor: 4.849

8.  Differential response to abiraterone acetate and di-n-butyl phthalate in an androgen-sensitive human fetal testis xenograft bioassay.

Authors:  Daniel J Spade; Susan J Hall; Camelia M Saffarini; Susan M Huse; Elizabeth V McDonnell; Kim Boekelheide
Journal:  Toxicol Sci       Date:  2013-11-27       Impact factor: 4.849

9.  Simvastatin and dipentyl phthalate lower ex vivo testicular testosterone production and exhibit additive effects on testicular testosterone and gene expression via distinct mechanistic pathways in the fetal rat.

Authors:  Brandiese E J Beverly; Christy S Lambright; Johnathan R Furr; Hunter Sampson; Vickie S Wilson; Barry S McIntyre; Paul M D Foster; Gregory Travlos; L Earl Gray
Journal:  Toxicol Sci       Date:  2014-07-23       Impact factor: 4.849

10.  Time- and dose-related effects of di-(2-ethylhexyl) phthalate and its main metabolites on the function of the rat fetal testis in vitro.

Authors:  François Chauvigné; Arnaud Menuet; Laurianne Lesné; Marie-Christine Chagnon; Cécile Chevrier; Jean-François Regnier; Jürgen Angerer; Bernard Jégou
Journal:  Environ Health Perspect       Date:  2008-12-01       Impact factor: 9.031

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