| Literature DB >> 15935386 |
E K Tan1, L L Chan, A P Auchus.
Abstract
Hemidystonia is frequently due to an underlying structural lesion in the basal ganglia and thalamus. It has been suggested that a preserved corticospinal tract may be required for hemidystonia to manifest. We provide the first report of a patient who presented with rapid-onset hemidystonia precipitated by an acute pontine infarct demonstrated on diffusion-weighted magnetic resonance imaging. Acute dysregulation of pallidal efferents to the pedunculopontine and/or pontine afferents to the thalamus may precipitate hemidystonia.Entities:
Mesh：
Year: 2005 PMID： 15935386 DOI： 10.1016/j.jns.2005.03.046
Source DB: PubMed Journal: J Neurol Sci ISSN： 0022-510X Impact factor: 3.181