| Literature DB >> 15793153 |
María T Andrés1, Mónica Viejo-Diaz, Francisco Pérez, José F Fierro.
Abstract
Lactoferrin-induced cell depolarization and a delayed tobramycin-killing effect on Pseudomonas aeruginosa cells were correlated. This antibiotic tolerance effect (ATE) reflects the ability of a defense protein to modify the activity of an antibiotic as a result of its modulatory effect on bacterial physiology. P. aeruginosa isolates from cystic fibrosis patients showed higher ATE values (< or = 6-fold) than other clinical strains.Entities:
Mesh:
Substances:
Year: 2005 PMID: 15793153 PMCID: PMC1068597 DOI: 10.1128/AAC.49.4.1613-1616.2005
Source DB: PubMed Journal: Antimicrob Agents Chemother ISSN: 0066-4804 Impact factor: 5.191