Literature DB >> 15784030

Increased acylation stimulating protein concentrations in nonalcoholic fatty liver disease are associated with insulin resistance.

Zeki Yesilova1, Metin Ozata, Cagatay Oktenli, Sait Bagci, Ayhan Ozcan, S Yavuz Sanisoglu, Ahmet Uygun, Halil Yaman, Necmettin Karaeren, Kemal Dagalp.   

Abstract

OBJECTIVES: As acylation stimulating protein (ASP) acts on adipocytes mainly as a paracrine factor to increase triglyceride synthesis and storage; hypothetically, it may play a similar role in the pathogenesis of nonalcoholic fatty liver disease (NAFLD).
METHODS: Forty-six male patients with NAFLD (group A), age-matched 30 male patients with chronic viral hepatitis (group B) and 30 age-matched and body mass index (BMI)-matched healthy male subjects were enrolled in the study.
RESULTS: Among the NAFLD patients, 10 patients (24.4%) had simple steatosis and 36 patients (69.6%) had nonalcoholic steatohepatitis (NASH). The mean levels of ASP, complement 3, insulin, C-peptide, HOMA-IR, triglyceride, and very low-density lipoprotein (VLDL) were significantly higher in group A patients than both controls and group B. ASP levels correlated significantly in a positive manner with BMI, insulin, and HOMA-IR.
CONCLUSIONS: Dysregulation of the ASP pathway may have important metabolic consequences in NASH and is associated with insulin resistance.

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Year:  2005        PMID: 15784030     DOI: 10.1111/j.1572-0241.2005.40838.x

Source DB:  PubMed          Journal:  Am J Gastroenterol        ISSN: 0002-9270            Impact factor:   10.864


  11 in total

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