Literature DB >> 15735156

CagA in Barrett's oesophagus in Colombia, a country with a high prevalence of gastric cancer.

M Kudo1, O Gutierrez, H M T El-Zimaity, H Cardona, Z Z Nurgalieva, J Wu, D Y Graham.   

Abstract

BACKGROUND: In the USA, atrophic gastritis and gastric cancer are rare, whereas gastro-oesophageal reflux disease (GERD) is common. Infection with Helicobacter pylori, especially a CagA positive strain, is unusual in patients with GERD/Barrett's oesophagus in the USA. AIM: To examine the relation between Barrett's oesophagus and CagA positive H pylori in Colombia, a country with a high prevalence of CagA positive H pylori associated atrophic gastritis and gastric cancer.
METHODS: Helicobacter pylori and CagA status was determined among Colombian patients with long segment Barrett's oesophagus and a control group with simple H pylori gastritis. Helicobacter pylori status was determined using a triple stain and CagA status was determined by immunohistochemistry using a specific rabbit anti-CagA serum.
RESULTS: Gastric and oesophageal mucosal biopsies were obtained from 51 patients--39 men (mean age, 57.8 years; SD, 13.1) and 12 women (mean age, 51.8 years; SD, 14.4)--with documented long segment Barrett's oesophagus. The results were compared with 24 Colombian patients with H pylori gastritis without oesophageal disease. Thirty two patients with Barrett's oesophagus had active H pylori infection. CagA status was evaluated in a subset of 23 H pylori infected patients with Barrett's oesophagus, and was positive in eight of these patients compared with 19 of 24 controls (p = 0.01).
CONCLUSIONS: Although most Colombian patients with Barrett's oesophagus had H pylori infection, CagA positive infections were unusual. These data illustrate how consistent corpus inflammation reduces acid secretion, which prevents Barrett's oesophagus among those with abnormal gastro-oesophageal reflux barriers.

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Year:  2005        PMID: 15735156      PMCID: PMC1770586          DOI: 10.1136/jcp.2004.022251

Source DB:  PubMed          Journal:  J Clin Pathol        ISSN: 0021-9746            Impact factor:   3.411


  44 in total

1.  Prospective evaluation of the prevalence of gastric Helicobacter pylori infection in patients with GERD, Barrett's esophagus, Barrett's dysplasia, and Barrett's adenocarcinoma.

Authors:  A P Weston; A S Badr; M Topalovski; R Cherian; A Dixon; R S Hassanein
Journal:  Am J Gastroenterol       Date:  2000-02       Impact factor: 10.864

2.  Modified triple stain (carbol fuchsin/alcian blue/hematoxylin-eosin) for the identification of Helicobacter pylori.

Authors:  H M El-Zimaity
Journal:  Arch Pathol Lab Med       Date:  2000-10       Impact factor: 5.534

3.  The role of interleukin-1 polymorphisms in the pathogenesis of gastric cancer.

Authors:  E M El-Omar; M Carrington; W H Chow; K E McColl; J H Bream; H A Young; J Herrera; J Lissowska; C C Yuan; N Rothman; G Lanyon; M Martin; J F Fraumeni; C S Rabkin
Journal:  Nature       Date:  2001-07-05       Impact factor: 49.962

4.  The phenotype of gastric mucosa coexisting with Barrett's oesophagus.

Authors:  M Rugge; V Russo; G Busatto; R M Genta; F Di Mario; F Farinati; D Y Graham
Journal:  J Clin Pathol       Date:  2001-06       Impact factor: 3.411

5.  Colonization with cagA-positive Helicobacter pylori strains inversely associated with reflux esophagitis and Barrett's esophagus.

Authors:  R J Loffeld; B F Werdmuller; J G Kuster; G I Pérez-Pérez; M J Blaser; E J Kuipers
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6.  Helicobacter pylori genotypes may determine gastric histopathology.

Authors:  C Nogueira; C Figueiredo; F Carneiro; A T Gomes; R Barreira; P Figueira; C Salgado; L Belo; A Peixoto; J C Bravo; L E Bravo; J L Realpe; A P Plaisier; W G Quint; B Ruiz; P Correa; L J van Doorn
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7.  Molecular epidemiology of Helicobacter pylori: separation of H. pylori from East Asian and non-Asian countries.

Authors:  Y Yamaoka; M S Osato; A R Sepulveda; O Gutierrez; N Figura; J G Kim; T Kodama; K Kashima; D Y Graham
Journal:  Epidemiol Infect       Date:  2000-02       Impact factor: 2.451

8.  Polymorphisms of two fucosyltransferase genes (Lewis and Secretor genes) involving type I Lewis antigens are associated with the presence of anti-Helicobacter pylori IgG antibody.

Authors:  Y Ikehara; S Nishihara; H Yasutomi; T Kitamura; K Matsuo; N Shimizu; K Inada; Y Kodera; Y Yamamura; H Narimatsu; N Hamajima; M Tatematsu
Journal:  Cancer Epidemiol Biomarkers Prev       Date:  2001-09       Impact factor: 4.254

9.  CagA-positive strains of Helicobacter pylori may protect against Barrett's esophagus.

Authors:  M F Vaezi; G W Falk; R M Peek; J J Vicari; J R Goldblum; G I Perez-Perez; T W Rice; M J Blaser; J E Richter
Journal:  Am J Gastroenterol       Date:  2000-09       Impact factor: 10.864

10.  Epidemiological differences between adenocarcinoma of the oesophagus and adenocarcinoma of the gastric cardia in the USA.

Authors:  H B El-Serag; A C Mason; N Petersen; C R Key
Journal:  Gut       Date:  2002-03       Impact factor: 23.059

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  2 in total

1.  CagA positivity and its association with gastroduodenal disease in Turkish children undergoing endoscopic investigation.

Authors:  Semra Sökücü; Ayşe Tülin Ozden; Ozlem Durmaz Süoğlu; Berna Elkabes; Fikri Demir; Uğur Cevikbaş; Selim Gökçe; Günay Saner
Journal:  J Gastroenterol       Date:  2006-06       Impact factor: 7.527

2.  Helicobacter pylori infection is associated with reduced risk of Barrett's esophagus: a meta-analysis and systematic review.

Authors:  Yan-Lin Du; Ru-Qiao Duan; Li-Ping Duan
Journal:  BMC Gastroenterol       Date:  2021-12-07       Impact factor: 3.067

  2 in total

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