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Literature DB >> 15721234

Minis: whence and wherefore?

Abstract

In this issue of Neuron, Sara et al. find that spontaneously released miniature synaptic potentials arise from a pool of vesicles distinct from those released by neural activity. This modification of a basic tenet of the quantal hypothesis has important implications for the analysis of changes in synaptic transmission.

Entities: Gene

Mesh：

Year: 2005 PMID： 15721234 DOI： 10.1016/j.neuron.2005.02.003

Source DB: PubMed Journal: Neuron ISSN： 0896-6273 Impact factor: 17.173

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Cited

20 in total

1. Post-tetanic potentiation is caused by two signalling mechanisms affecting quantal size and quantal content.

Authors: Lei Xue; Ling-Gang Wu
Journal: J Physiol Date: 2010-11-01 Impact factor: 5.182

Review 2. Leaky synapses: regulation of spontaneous neurotransmission in central synapses.

Authors: C R Wasser; E T Kavalali
Journal: Neuroscience Date: 2008-03-22 Impact factor: 3.590

3. The Long 3'UTR mRNA of CaMKII Is Essential for Translation-Dependent Plasticity of Spontaneous Release in Drosophila melanogaster.

Authors: Elena A Kuklin; Stephen Alkins; Baskar Bakthavachalu; Maria C Genco; Indulekha Sudhakaran; K Vijay Raghavan; Mani Ramaswami; Leslie C Griffith
Journal: J Neurosci Date: 2017-09-27 Impact factor: 6.167

10. Corticostriatal synaptic function in mouse models of Huntington's disease: early effects of huntingtin repeat length and protein load.

Authors: Austen J Milnerwood; Lynn A Raymond
Journal: J Physiol Date: 2007-10-18 Impact factor: 5.182

Minis: whence and wherefore?

1. Post-tetanic potentiation is caused by two signalling mechanisms affecting quantal size and quantal content.

Review 2. Leaky synapses: regulation of spontaneous neurotransmission in central synapses.

3. The Long 3'UTR mRNA of CaMKII Is Essential for Translation-Dependent Plasticity of Spontaneous Release in Drosophila melanogaster.

4. Evoked and spontaneous transmission favored by distinct sets of synapses.

5. Synaptic neuropeptide release induced by octopamine without Ca2+ entry into the nerve terminal.

6. Increased expression of DNA methyltransferase 1 and 3a in human temporal lobe epilepsy.

7. Spontaneous and evoked glutamate release activates two populations of NMDA receptors with limited overlap.

8. Rapid activity-dependent modifications in synaptic structure and function require bidirectional Wnt signaling.

9. Calcium regulation of spontaneous and asynchronous neurotransmitter release.

10. Corticostriatal synaptic function in mouse models of Huntington's disease: early effects of huntingtin repeat length and protein load.