Literature DB >> 15643916

Oral clefts, maternal smoking, and TGFA: a meta-analysis of gene-environment interaction.

Joanna S Zeiger1, Terri H Beaty, Kung-Yee Liang.   

Abstract

OBJECTIVE: A meta-analysis was performed to examine the association among maternal cigarette smoking, infant genotype at the Taq1 site in the transforming growth factor alpha (TGFA) locus, and risk of nonsyndromic oral clefts, both cleft palate (CP) and cleft lip with or without cleft palate (CL/P).
DESIGN: Five published case-control studies were included in the meta-analyis. Pooled Mantel-Haenszel odds ratios (OR) and 95% confidence intervals (CIs) were computed. Gene-environment interaction was also assessed by using the pooled data in a case-only analysis and polytomous logistic regression.
RESULTS: Among nonsmoking mothers, there was no evidence of any increased risk for CP if the infant carried the TGFA Taq1 C2 allele. If the mother reported smoking, however, there was an overall increased risk for CP if the infant carried the C2 allele (ORsmokers = 1.95; 95% CI = 1.22 to 3.10). TGFA genotype did not increase risk to CL/P, regardless of maternal smoking status. Polytomous logistic regression revealed a significant overall smoking effect for CL/P (OR = 1.64, 95% CI = 1.33 to 2.02) and CP (OR = 1.42, 95% CI = 1.06 to 1.90).
CONCLUSIONS: While maternal smoking was a consistent risk factor for both CL/P and CP across all studies, the suggestive evidence for gene-environment interaction between the infant's genotype at the Taq1 marker in TGFA and maternal smoking was limited to CP. Furthermore, evidence for such gene-environment interaction was strongest in a case-control study drawn from a birth defect registry where infants with non-cleft defects served as controls.

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Year:  2005        PMID: 15643916     DOI: 10.1597/02-128.1

Source DB:  PubMed          Journal:  Cleft Palate Craniofac J        ISSN: 1055-6656


  31 in total

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Journal:  Cleft Palate Craniofac J       Date:  2011-11-10

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Review 3.  Genetics of nonsyndromic orofacial clefts.

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Journal:  Cleft Palate Craniofac J       Date:  2011-05-05

4.  Evidence for gene-environment interaction in a genome wide study of nonsyndromic cleft palate.

Authors:  Terri H Beaty; Ingo Ruczinski; Jeffrey C Murray; Mary L Marazita; Ronald G Munger; Jacqueline B Hetmanski; Tanda Murray; Richard J Redett; M Daniele Fallin; Kung Yee Liang; Tao Wu; Poorav J Patel; Sheng-Chih Jin; Tian Xiao Zhang; Holger Schwender; Yah Huei Wu-Chou; Philip K Chen; Samuel S Chong; Felicia Cheah; Vincent Yeow; Xiaoqian Ye; Hong Wang; Shangzhi Huang; Ethylin W Jabs; Bing Shi; Allen J Wilcox; Rolv T Lie; Sun Ha Jee; Kaare Christensen; Kimberley F Doheny; Elizabeth W Pugh; Hua Ling; Alan F Scott
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5.  Identifying Human Teratogens: An Update.

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6.  Genetic variants in tooth agenesis-related genes might be also involved in tooth size variations.

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Review 7.  Environmental exposures and development.

Authors:  Donald R Mattison
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Review 8.  Review on genetic variants and maternal smoking in the etiology of oral clefts and other birth defects.

Authors:  Min Shi; George L Wehby; Jeffrey C Murray
Journal:  Birth Defects Res C Embryo Today       Date:  2008-03

9.  Evidence that TGFA influences risk to cleft lip with/without cleft palate through unconventional genetic mechanisms.

Authors:  Jae Woong Sull; Kung-Yee Liang; Jacqueline B Hetmanski; Tao Wu; Margaret Daniele Fallin; Roxann G Ingersoll; Ji Wan Park; Yah-Huei Wu-Chou; Philip K Chen; Samuel S Chong; Felicia Cheah; Vincent Yeow; Beyoung Yun Park; Sun Ha Jee; Ethylin Wang Jabs; Richard Redett; Alan F Scott; Terri H Beaty
Journal:  Hum Genet       Date:  2009-05-15       Impact factor: 4.132

10.  Addressing the challenges of cleft lip and palate research in India.

Authors:  Peter Mossey; Julian Little
Journal:  Indian J Plast Surg       Date:  2009-10
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