Literature DB >> 15607315

A novel endogenous digitalis, telocinobufagin, exhibits elevated plasma levels in patients with terminal renal failure.

Yutaka Komiyama1, Xian Hui Dong, Noriko Nishimura, Hiroya Masaki, Masamichi Yoshika, Midori Masuda, Hakuo Takahashi.   

Abstract

BACKGROUND AND OBJECTIVES: There are several potential endogenous digitalis-like factors (EDLF) in mammalian body fluids, and marinobufagenin (MBG) may be the most potent EDLF. Improved assays are needed to confirm the potency of these metabolites. In the present study, we have identified MBG and telocinobufagin (TCB) in human plasma by high-resolution mass spectrometry (MS) and nuclear magnetic resonance (NMR). METHODS AND
RESULTS: The high-resolution MS analysis revealed the molecular masses of TCB and MBG to be the same as their respective theoretical values. Using a tandem mass spectrometer, the mass-charge ratio for TCB was determined to be 403.2 for the parent ion and 349.2 for the daughter ion. The mass-charge ratio for MBG was m/z 383.2 and m/z 401.2. The NMR study revealed that the signals for MBG and TCB were the same as those obtained by MS analysis. In human blood, MBG and TCB were also identified by liquid chromatography (LC) as well as MS. In the LC/MS assay, proscillaridin A was used as an internal standard. The plasma was pretreated with Sep-Pak C18, and then 50 microL was applied to the C8 high-performance liquid chromatography (HPLC) column. The mean plasma concentration of MBG in healthy volunteers (0.94 +/- 0.28 ng/mL) was significantly lower than that in patients undergoing regular hemodialysis (3.81 +/- 1.92 ng/mL). The concentration of TCB in the healthy volunteers (1.80 +/- 0.55 ng/mL) was also significantly lower than that in patients with terminal renal failure (6.86 +/- 4.30 ng/mL).
CONCLUSION: These results indicate that the major EDLF is TCB because its plasma concentration is the highest among the reported endogenous digitalis candidates.

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Year:  2005        PMID: 15607315     DOI: 10.1016/j.clinbiochem.2004.08.005

Source DB:  PubMed          Journal:  Clin Biochem        ISSN: 0009-9120            Impact factor:   3.281


  52 in total

1.  Reduction of Na/K-ATPase potentiates marinobufagenin-induced cardiac dysfunction and myocyte apoptosis.

Authors:  Changxuan Liu; Yan Bai; Yiliang Chen; Yu Wang; Yoann Sottejeau; Lijun Liu; Xiaomei Li; Jerry B Lingrel; Deepak Malhotra; Christopher J Cooper; Joseph I Shapiro; Zi-jian Xie; Jiang Tian
Journal:  J Biol Chem       Date:  2012-03-27       Impact factor: 5.157

2.  Proinflammatory Effects of Cardiotonic Steroids Mediated by NKA α-1 (Na+/K+-ATPase α-1)/Src Complex in Renal Epithelial Cells and Immune Cells.

Authors:  Fatimah K Khalaf; Prabhatchandra Dube; Andrew L Kleinhenz; Deepak Malhotra; Amira Gohara; Christopher A Drummond; Jiang Tian; Steven T Haller; Zijian Xie; David J Kennedy
Journal:  Hypertension       Date:  2019-05-28       Impact factor: 10.190

3.  Elevated Plasma Marinobufagenin, An Endogenous Cardiotonic Steroid, Is Associated With Right Ventricular Dysfunction and Nitrative Stress in Heart Failure.

Authors:  David J Kennedy; Kevin Shrestha; Brendan Sheehey; Xinmin S Li; Anuradha Guggilam; Yuping Wu; Michael Finucan; Alaa Gabi; Charles M Medert; Kristen Westfall; Allen Borowski; Olga Fedorova; Alexei Y Bagrov; W H Wilson Tang
Journal:  Circ Heart Fail       Date:  2015-08-14       Impact factor: 8.790

Review 4.  The Na/K-ATPase/Src complex and cardiotonic steroid-activated protein kinase cascades.

Authors:  Zhichuan Li; Zijian Xie
Journal:  Pflugers Arch       Date:  2008-02-19       Impact factor: 3.657

Review 5.  Endogenous digitalis: pathophysiologic roles and therapeutic applications.

Authors:  Alexei Y Bagrov; Joseph I Shapiro
Journal:  Nat Clin Pract Nephrol       Date:  2008-06-10

Review 6.  Endogenous cardiotonic steroids: physiology, pharmacology, and novel therapeutic targets.

Authors:  Alexei Y Bagrov; Joseph I Shapiro; Olga V Fedorova
Journal:  Pharmacol Rev       Date:  2009-03       Impact factor: 25.468

Review 7.  Endogenous cardiotonic steroids in kidney failure: a review and an hypothesis.

Authors:  John M Hamlyn; Paolo Manunta
Journal:  Adv Chronic Kidney Dis       Date:  2015-05       Impact factor: 3.620

Review 8.  Regulation of renal function and structure by the signaling Na/K-ATPase.

Authors:  Jeffrey X Xie; Xin Li; Zijian Xie
Journal:  IUBMB Life       Date:  2013-12-10       Impact factor: 3.885

9.  The cardiotonic steroid hormone marinobufagenin induces renal fibrosis: implication of epithelial-to-mesenchymal transition.

Authors:  Larisa V Fedorova; Vanamala Raju; Nasser El-Okdi; Amjad Shidyak; David J Kennedy; Sandeep Vetteth; David R Giovannucci; Alexei Y Bagrov; Olga V Fedorova; Joseph I Shapiro; Deepak Malhotra
Journal:  Am J Physiol Renal Physiol       Date:  2009-01-28

10.  Cinobufotalin impedes Sw.71 cytotrophoblast cell line function via cell cycle arrest and apoptotic signaling.

Authors:  Syeda H Afroze; Jenna Sloan; Grace-Ann C Osuji; Nathan Drever; Kimberly Pilkinton; David C Zawieja; Thomas J Kuehl; M Nasir Uddin
Journal:  Mol Cell Biochem       Date:  2016-10-03       Impact factor: 3.396

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