Literature DB >> 15567755

Influence of norcantharidin on proliferation, proliferation-related gene proteins proliferating cell nuclear antigen and Ki-67 of human gallbladder carcinoma GBC-SD cells.

Yue-Zu Fan1, Jin-Ye Fu, Ze-Ming Zhao, Cun-Qiu Chen.   

Abstract

BACKGROUND: Gallbladder carcinoma is a highly lethal and aggressive disease with early metastasis, strong invasion and poor prognosis. Most patients with this disease are at the advanced and un-resectable stage and should be considered for palliative treatment such as chemotherapy and radiotherapy. Unfortunately, reports of chemotherapy and radiotherapy for gallbladder carcinoma are disappointing. We investigated the influence of norcantharidin (NCTD) on proliferation, proliferation-related gene proteins PCNA and Ki-67 of human gallbladder carcinoma GBC-SD cells in vitro.
METHODS: GBC-SD cell lines of human gallbladder carcinoma were cultured by the cell culture technique. The experiment was divided into NCTD group and control group. The tetrazolium-based colorimetric assay was used to evaluate cell growth. The streptavidin-biotin complex method was used to determine the expressions of proliferation-related gene proteins PCNA and Ki-67 of human gallbladder carcinoma GBC-SD cells.
RESULTS: NCTD inhibited the growth and proliferation of GBC-SD cells from 10 mg/L or after 6 hours in a dose-and time-dependent manner, with the IC50 value of 56.18 microg/ml at 48 hours. After treatment with NCTD, the expression of PCNA (0.932+/-0.031 vs. 0.318+/-0.023, P<0.001) and Ki-67 (0.964+/-0.092 vs. 0.297+/-0.018, P<0.001) proteins were decreased significantly.
CONCLUSION: NCTD inhibits the proliferation of human gallbladder carcinoma GBC-SD cells in vitro and the expression of their proliferation-related gene proteins PCNA and Ki-67.

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Year:  2004        PMID: 15567755

Source DB:  PubMed          Journal:  Hepatobiliary Pancreat Dis Int


  5 in total

1.  Interferon-gamma and interlukin-4 patterns in BALB/c mice suffering from cutaneous leishmaniasis treated with cantharidin.

Authors:  Yahya Maroufi; Fatemeh Ghaffarifar; Abdolhosein Dalimi; Zohreh Sharifi
Journal:  Jundishapur J Microbiol       Date:  2014-06-01       Impact factor: 0.747

2.  Norcantharidin induced DU145 cell apoptosis through ROS-mediated mitochondrial dysfunction and energy depletion.

Authors:  Bo Shen; Pei-Jie He; Chun-Lin Shao
Journal:  PLoS One       Date:  2013-12-19       Impact factor: 3.240

3.  Norcantharidin Suppresses Colon Cancer Cell Epithelial-Mesenchymal Transition by Inhibiting the αvβ6-ERK-Ets1 Signaling Pathway.

Authors:  Cheng Peng; Zequn Li; Zhengchuan Niu; Wei Niu; Zongquan Xu; Huijie Gao; Weibo Niu; JiaYong Wang; Zhaobin He; Chao Gao; Pengfei Lin; Michael Agrez; Zongli Zhang; Jun Niu
Journal:  Sci Rep       Date:  2016-02-05       Impact factor: 4.379

4.  DMH1 (4-[6-(4-isopropoxyphenyl)pyrazolo[1,5-a]pyrimidin-3-yl]quinoline) inhibits chemotherapeutic drug-induced autophagy.

Authors:  Yue Sheng; Bo Sun; Xin Xie; Na Li; Deli Dong
Journal:  Acta Pharm Sin B       Date:  2015-02-21       Impact factor: 11.413

Review 5.  Insight into norcantharidin, a small-molecule synthetic compound with potential multi-target anticancer activities.

Authors:  Mu-Su Pan; Jin Cao; Yue-Zu Fan
Journal:  Chin Med       Date:  2020-05-29       Impact factor: 5.455

  5 in total

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