Literature DB >> 15539147

Fingering IL-12 with apoptotic cells.

Peter M Henson1.   

Abstract

Exposure of phosphatidylserine on the surface of apoptotic cells contributes to their removal in situ by quiet, noninflammatory, and nonimmunogenic mechanisms. In this issue of Immunity, describe a completely new mechanism for this effect that involves a novel zinc finger nuclear factor that inhibits IL-12 production.

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Year:  2004        PMID： 15539147     DOI： 10.1016/j.immuni.2004.11.002

Source DB:  PubMed          Journal:  Immunity        ISSN： 1074-7613            Impact factor:   31.745


  4 in total

1.  Leishmania disease development depends on the presence of apoptotic promastigotes in the virulent inoculum.

Authors:  Ger van Zandbergen; Annalena Bollinger; Alexander Wenzel; Shaden Kamhawi; Reinhard Voll; Matthias Klinger; Antje Müller; Christoph Hölscher; Martin Herrmann; David Sacks; Werner Solbach; Tamás Laskay
Journal:  Proc Natl Acad Sci U S A       Date:  2006-08-31       Impact factor: 11.205

2.  Annexin V expression on CD4+ T cells with regulatory function.

Authors:  Anna-Lena Bollinger; Thomas Bollinger; Jan Rupp; Kensuke Shima; Natalie Gross; Laura Padayachy; Rachel Chicheportiche; Gisella L Puga Yung; Jörg Dieter Seebach
Journal:  Immunology       Date:  2019-11-20       Impact factor: 7.397

3.  Ascorbate peroxidase from Leishmania major controls the virulence of infective stage of promastigotes by regulating oxidative stress.

Authors:  Swati Pal; Subhankar Dolai; Rajesh K Yadav; Subrata Adak
Journal:  PLoS One       Date:  2010-06-23       Impact factor: 3.240

4.  Subversion of Immunity by Leishmania amazonensis Parasites: Possible Role of Phosphatidylserine as a Main Regulator.

Authors:  Joao Luiz Mendes Wanderley; Jaqueline França Costa; Valéria Matos Borges; Marcello Barcinski
Journal:  J Parasitol Res       Date:  2012-02-02
  4 in total

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