Literature DB >> 15505122

A meta-analysis comparing the effect of thiazolidinediones on cardiovascular risk factors.

Elaine Chiquette1, Gilbert Ramirez, Ralph Defronzo.   

Abstract

BACKGROUND: In patients with type 2 diabetes mellitus, all therapeutic options should be evaluated for their effect on cardiovascular risk factors, in addition to glycemic control. We conducted a meta-analysis of randomized controlled trials of pioglitazone hydrochloride and rosiglitazone maleate in patients with type 2 diabetes to evaluate their effect on glycemic control, lipids, blood pressure, and weight.
METHODS: Randomized controlled trials of patients with type 2 diabetes that compared pioglitazone or rosiglitazone with placebo for 12 weeks were included. Primary analysis was to compare thiazolidinediones with placebo. Secondary analysis was to identify whether treatment with pioglitazone differed from rosiglitazone in any outcomes. We calculated weighted mean differences and 95% confidence intervals.
RESULTS: Twenty-three randomized controlled trials were identified. Both thiazolidinediones demonstrated similar hemoglobin A(1c) level decreases of 1.0% to 1.5% and similar increases in body weight of approximately 3.0 kg. Pioglitazone significantly lowered triglyceride level (-40 mg/dL [-0.45 mmol/L]; 95% confidence interval [CI], -53 to -26 mg/dL [-0.60 to -0.29 mmol/L]), increased high-density lipoprotein cholesterol (HDL-C) level (+4.6 mg/dL [+0.12 mmol/L]; 95% CI, 3.6 to 5.5 mg/dL [0.09 to 0.14 mmol/L]), and showed neutral effect on low-density lipoprotein cholesterol (LDL-C) and total cholesterol levels. Rosiglitazone significantly increased HDL-C level (+2.7 mg/dL [+0.07 mmol/L]; 95% CI, 2.0 to 3.4 mg/dL [0.05 to 0.09 mmol/L]), but increased LDL-C level (+15 mg/dL [+0.39 mmol/L]; 95% CI, 13 to 17 mg/dL [0.34 to 0.44 mmol/L]), total cholesterol level (+21 mg/dL [+0.54 mmol/L]; 95% CI, 18 to 25 mg/dL [0.47 to 0.65 mmol/L]), and demonstrated neutral effect on triglyceride level (-1.1 mg/dL [-0.12 mmol/L]; 95% CI, -14 to 12 mg/dL [-0.16 to 0.14 mmol/L]). No data were available on pioglitazone and blood pressure. Rosiglitazone had a neutral effect on systolic (-0.7 mm Hg; 95% CI, -2.6 to 1.1 mm Hg) and diastolic (-0.8 mm Hg; 95% CI, -1.8 to 0.3) blood pressure.
CONCLUSIONS: Thiazolidinediones have similar effects on glycemic control and body weight. Pioglitazone produced a more favorable lipid profile. Head-to-head comparative trials as well as longer-term cardiovascular outcome studies are needed to determine whether there are differences in efficacy between the 2 thiazolidinediones.

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Year:  2004        PMID: 15505122     DOI: 10.1001/archinte.164.19.2097

Source DB:  PubMed          Journal:  Arch Intern Med        ISSN: 0003-9926


  66 in total

1.  Short-term continuous subcutaneous insulin infusion combined with insulin sensitizers rosiglitazone, metformin, or antioxidant α-lipoic acid in patients with newly diagnosed type 2 diabetes mellitus.

Authors:  Zhimin Huang; Xuesi Wan; Juan Liu; Wanping Deng; Ailing Chen; Liehua Liu; Jianbin Liu; Guohong Wei; Hai Li; Donghong Fang; Yanbing Li
Journal:  Diabetes Technol Ther       Date:  2013-08-30       Impact factor: 6.118

Review 2.  Pharmacokinetic interactions with thiazolidinediones.

Authors:  André J Scheen
Journal:  Clin Pharmacokinet       Date:  2007       Impact factor: 6.447

3.  Effect of rosiglitazone on HDL metabolism in subjects with metabolic syndrome and low HDL.

Authors:  John S Millar; Katsunori Ikewaki; LeAnne T Bloedon; Megan L Wolfe; Philippe O Szapary; Daniel J Rader
Journal:  J Lipid Res       Date:  2010-10-22       Impact factor: 5.922

4.  Rosiglitazone inhibits acyl-CoA synthetase activity and fatty acid partitioning to diacylglycerol and triacylglycerol via a peroxisome proliferator-activated receptor-gamma-independent mechanism in human arterial smooth muscle cells and macrophages.

Authors:  Bardia Askari; Jenny E Kanter; Ashley M Sherrid; Deidre L Golej; Andrew T Bender; Joey Liu; Willa A Hsueh; Joseph A Beavo; Rosalind A Coleman; Karin E Bornfeldt
Journal:  Diabetes       Date:  2007-01-26       Impact factor: 9.461

Review 5.  Exploration and Development of PPAR Modulators in Health and Disease: An Update of Clinical Evidence.

Authors:  Hong Sheng Cheng; Wei Ren Tan; Zun Siong Low; Charlie Marvalim; Justin Yin Hao Lee; Nguan Soon Tan
Journal:  Int J Mol Sci       Date:  2019-10-11       Impact factor: 5.923

Review 6.  Insulin Resistance and Atherosclerosis: Implications for Insulin-Sensitizing Agents.

Authors:  Antonino Di Pino; Ralph A DeFronzo
Journal:  Endocr Rev       Date:  2019-12-01       Impact factor: 19.871

Review 7.  Effect of glitazones on the progression of coronary artery disease in type 2 diabetes patients.

Authors:  Jamison Wyatt; Shailesh Nandish; Rene Oliveros; Adrienne S Zion; Michael S Lujan; Robert Chilton
Journal:  Vasc Health Risk Manag       Date:  2010-02-04

8.  Defective peroxisomal proliferators activated receptor gamma activity due to dominant-negative mutation synergizes with hypertension to accelerate cardiac fibrosis in mice.

Authors:  Adrienn Kis; Colin Murdoch; Min Zhang; Anjana Siva; Sergio Rodriguez-Cuenca; Stefania Carobbio; Agnes Lukasik; Margaret Blount; Steve O'Rahilly; Sarah L Gray; Ajay M Shah; Antonio Vidal-Puig
Journal:  Eur J Heart Fail       Date:  2009-04-24       Impact factor: 15.534

9.  Peroxisome proliferator-activated receptor agonists: do they increase cardiovascular risk?

Authors:  Ahmad Aljada; Kshitij Ashwin Shah; Shaker A Mousa
Journal:  PPAR Res       Date:  2009-08-19       Impact factor: 4.964

Review 10.  Is there evidence that oral hypoglycemic agents reduce cardiovascular morbidity/mortality? Yes.

Authors:  Cristina Bianchi; Roberto Miccoli; Giuseppe Daniele; Giuseppe Penno; Stefano Del Prato
Journal:  Diabetes Care       Date:  2009-11       Impact factor: 19.112

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