Literature DB >> 1537380

Catalase inhibits nitric oxide synthesis and the killing of intracellular Leishmania major in murine macrophages.

Y Li1, A Severn, M V Rogers, R M Palmer, S Moncada, F Y Liew.   

Abstract

Mouse peritoneal macrophages activated with interferon-gamma (IFN-gamma) and lipopolysaccharide produce substantial amounts of nitric oxide (NO), which correlates with the elimination of the intracellular protozoan parasite Leishmania major. Both the production of NO and the leishmanicidal function of the activated macrophages can be significantly inhibited by catalase in a dose- and time-dependent manner. These results could not be interpreted by the reduction of H2O2 by catalase since the removal of H2O2 by the addition of glutathione peroxidase had no effect on the NO synthesis or the leishmanicidal function of activated macrophages. Furthermore, catalase did not affect the induction of NO synthase in IFN-gamma-activated macrophages. In contrast, the inhibition of NO synthesis and leishmanicidal activity by catalase was reversed in a dose-dependent manner by the addition of tetrahydrobiopterin, a cofactor of NO synthase. Taken together, these results not only further support the central role of NO as the cytotoxic moiety, but also suggest that hydrogen peroxide may interfere with NO production by affecting the levels of cofactor needed for its synthesis.

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Year:  1992        PMID: 1537380     DOI: 10.1002/eji.1830220223

Source DB:  PubMed          Journal:  Eur J Immunol        ISSN: 0014-2980            Impact factor:   5.532


  12 in total

1.  Comparative roles of free fatty acids with reactive nitrogen intermediates and reactive oxygen intermediates in expression of the anti-microbial activity of macrophages against Mycobacterium tuberculosis.

Authors:  T Akaki; H Tomioka; T Shimizu; S Dekio; K Sato
Journal:  Clin Exp Immunol       Date:  2000-08       Impact factor: 4.330

2.  Protection of islet cells from inflammatory cell death in vitro.

Authors:  V Burkart; H Kolb
Journal:  Clin Exp Immunol       Date:  1993-08       Impact factor: 4.330

3.  Erythrocyte oxidant/antioxidant status in essential hyperhidrosis.

Authors:  Semsettin Karaca; Mustafa Kulac; Efkan Uz; Hakan Mollaoglu; H Ramazan Yilmaz
Journal:  Mol Cell Biochem       Date:  2006-05-23       Impact factor: 3.396

4.  A commercial preparation of catalase inhibits nitric oxide production by activated murine macrophages: role of arginase.

Authors:  Y Tian; Y Xing; R Magliozzo; K Yu; B R Bloom; J Chan
Journal:  Infect Immun       Date:  2000-05       Impact factor: 3.441

5.  Inhibition of macrophage nitric oxide production by arachidonate-cascade inhibitors.

Authors:  K Ryoyama; T Nomura; S Nakamura
Journal:  Cancer Immunol Immunother       Date:  1993-11       Impact factor: 6.968

6.  Lack of involvement of nitric oxide in killing of Aspergillus fumigatus conidia by pulmonary alveolar macrophages.

Authors:  E Michaliszyn; S Sénéchal; P Martel; L de Repentigny
Journal:  Infect Immun       Date:  1995-05       Impact factor: 3.441

7.  Overexpression of catalase delays G0/G1- to S-phase transition during cell cycle progression in mouse aortic endothelial cells.

Authors:  Ogbeyalu E Onumah; George E Jules; Yanfeng Zhao; LiChun Zhou; Hong Yang; ZhongMao Guo
Journal:  Free Radic Biol Med       Date:  2009-03-31       Impact factor: 7.376

Review 8.  Role of nitric oxide in parasitic infections.

Authors:  S L James
Journal:  Microbiol Rev       Date:  1995-12

9.  Comparative analysis of the nitric oxide production by Leishmania sp.

Authors:  Marcelo Genestra; Wilson Jacinto Silva de Souza; Léa Cysne-Finkelstein; Leonor L Leon
Journal:  Med Microbiol Immunol       Date:  2003-06-21       Impact factor: 3.402

10.  Nitric oxide potentiates hydrogen peroxide-induced killing of Escherichia coli.

Authors:  R Pacelli; D A Wink; J A Cook; M C Krishna; W DeGraff; N Friedman; M Tsokos; A Samuni; J B Mitchell
Journal:  J Exp Med       Date:  1995-11-01       Impact factor: 14.307

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