Literature DB >> 15365719

Photoshop-based image analysis of canine articular cartilage after subchondral damage.

A Lahm1, M Uhl, H A Lehr, C Ihling, P C Kreuz, J Haberstroh.   

Abstract

INTRODUCTION: The validity of histopathological grading is a major problem in the assessment of articular cartilage. Calculating the cumulative strength of signal intensity of different stains gives information regarding the amount of proteoglycan, glycoproteins, etc. Using this system, we examined the medium-term effect of subchondral lesions on initially healthy articular cartilage.
MATERIALS AND METHODS: After cadaver studies, an animal model was created to produce pure subchondral damage without affecting the articular cartilage in 12 beagle dogs under MRI control. Quantification of the different stains was provided using a Photoshop-based image analysis (pixel analysis) with the histogram command 6 months after subchondral trauma.
RESULTS: FLASH 3D sequences revealed intact cartilage after impact in all cases. The best detection of subchondral fractures was achieved with fat-suppressed TIRM sequences. Semiquantitative image analysis showed changes in proteoglycan and glycoprotein quantities in 9 of 12 samples that had not shown any evidence of damage during the initial examination. Correlation analysis showed a loss of the physiological distribution of proteoglycans and glycoproteins in the different zones of articular cartilage.
CONCLUSION: Currently available software programs can be applied for comparative analysis of histologic stains of hyaline cartilage. After subchondral fractures, significant changes in the cartilage itself occur after 6 months.

Entities:  

Mesh:

Substances:

Year:  2004        PMID: 15365719     DOI: 10.1007/s00402-004-0701-6

Source DB:  PubMed          Journal:  Arch Orthop Trauma Surg        ISSN: 0936-8051            Impact factor:   3.067


  7 in total

1.  Hypertrophic cervical elongation: clinical and histological correlations.

Authors:  Okechukwu A Ibeanu; Ralph R Chesson; Dane Sandquist; Javier Perez; Kerri Santiago; Thomas E Nolan
Journal:  Int Urogynecol J       Date:  2010-03-24       Impact factor: 2.894

2.  MiR-210 expression reverses radioresistance of stem-like cells of oesophageal squamous cell carcinoma.

Authors:  Xin Chen; Jia Guo; Ru-Xing Xi; Yu-Wei Chang; Fei-Yang Pan; Xiao-Zhi Zhang
Journal:  World J Clin Oncol       Date:  2014-12-10

3.  Crosstalk between the equilibrative nucleoside transporter ENT2 and alveolar Adora2b adenosine receptors dampens acute lung injury.

Authors:  Tobias Eckle; Kelly Hughes; Heidi Ehrentraut; Kelley S Brodsky; Peter Rosenberger; Doo-Sup Choi; Katya Ravid; Tingting Weng; Yang Xia; Michael R Blackburn; Holger K Eltzschig
Journal:  FASEB J       Date:  2013-04-19       Impact factor: 5.191

4.  Matrix metalloproteinases and their inhibitors in the chamber angle of normal eyes and patients with primary open-angle glaucoma and exfoliation glaucoma.

Authors:  Seppo Rönkkö; Petri Rekonen; Kai Kaarniranta; Tuomo Puustjärvi; Markku Teräsvirta; Hannu Uusitalo
Journal:  Graefes Arch Clin Exp Ophthalmol       Date:  2006-10-07       Impact factor: 3.535

5.  Quantitative immunohistochemistry by measuring chromogen signal strength using a C# written program.

Authors:  Shaghayegh Haghjooy Javanmard; Ali Moeiny
Journal:  J Res Med Sci       Date:  2009-05       Impact factor: 1.852

6.  A 3-Dimensional In Vitro Model of Zonally Organized Extracellular Matrix.

Authors:  Sonja M Walzer; Stefan Toegel; Catharina Chiari; Sebastian Farr; Beate Rinner; Annelie-Martina Weinberg; Daniela Weinmann; Michael B Fischer; Reinhard Windhager
Journal:  Cartilage       Date:  2019-08-02       Impact factor: 3.117

7.  Diurnal and nutritional adjustments of intracellular Ca2+ release channels and Ca2+ ATPases associated with restricted feeding schedules in the rat liver.

Authors:  Adrián Báez-Ruiz; Karina Cázares-Gómez; Olivia Vázquez-Martínez; Raúl Aguilar-Roblero; Mauricio Díaz-Muñoz
Journal:  J Circadian Rhythms       Date:  2013-08-20
  7 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.