Literature DB >> 15362962

Mutations in the NSD1 gene in patients with Sotos syndrome associate with endocrine and paracrine alterations in the IGF system.

L De Boer1, H A Van Duyvenvoorde, E C Willemstein-Van Hove, C M Hoogerbrugge, J Van Doorn, J A Maassen, M Karperien, J M Wit.   

Abstract

OBJECTIVE: To investigate the effect of nuclear receptor Su-var, 3-9, enhancer of zeste, trithorax (SET) domain-containing protein 1 (NSD1) gene alteration in patients with Sotos syndrome on plasma IGFs and IGF-binding proteins (IGFBPs), as well as on the IGF/IGFBP system activity at the tissue level.
DESIGN: Twenty-nine patients suspected of Sotos syndrome were divided into two groups: patients with heterozygous deletions or mutations in the NSD1 gene (NSD1(+/-)) (n=11) and subjects without (NSD1(+/+)) (n=18). Plasma samples (n=29) and skin fibroblasts (n=23) were obtained. The results of both groups were compared and related to reference values.
METHODS: IGF-I, IGF-II, IGFBP-2, IGFBP-3, IGFBP-4 and IGFBP-6 levels were determined by RIAs. The mitogenic response of fibroblasts to IGFs was investigated by [methyl-(3)H]thymidine incorporation. IGFBP-3 levels in the culture media were measured by RIA. IGFBP-3 mRNA expression was determined by real time RT-PCR.
RESULTS: NSD1(+/-) patients showed significantly altered levels of IGF-I (mean-1.2 SDS), IGF-II (-1.2), IGFBP-3 (-1.7), IGFBP-4 (-0.4), IGFBP-2 (+0.8) and IGFBP-6 (+1.5). The NSD1(+/+) patients did not differ from the reference, with the exception of the mean IGFBP-3 level (-1.3). Basal proliferation and mitogenic response to IGFs was diminished in NSD1(+/-) fibroblasts compared with NSD1(+/+) (basal, P=0.02; IGF-I, P<0.001; IGF-II, P=0.02). Compared with control fibroblasts, only the mitogenic response was diminished (basal, P=0.07; IGF-I, P=0.04; IGF-II, P=0.04). A trend of higher IGFBP-3 secretion after IGF-I stimulation (P=0.09) and 3.5-5 times higher mRNA expression of IGFBP-3 in basal conditions was found in NSD1(+/-) fibroblasts in comparison to controls.
CONCLUSIONS: NSD1(+/-) patients show endocrine and paracrine changes in the IGF system. These changes may contribute to the abnormal growth pattern.

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Year:  2004        PMID: 15362962     DOI: 10.1530/eje.0.1510333

Source DB:  PubMed          Journal:  Eur J Endocrinol        ISSN: 0804-4643            Impact factor:   6.664


  5 in total

Review 1.  Malan syndrome: Sotos-like overgrowth with de novo NFIX sequence variants and deletions in six new patients and a review of the literature.

Authors:  Merel Klaassens; Deborah Morrogh; Elisabeth M Rosser; Fatima Jaffer; Maaike Vreeburg; Levinus A Bok; Tim Segboer; Martine van Belzen; Ros M Quinlivan; Ajith Kumar; Jane A Hurst; Richard H Scott
Journal:  Eur J Hum Genet       Date:  2014-08-13       Impact factor: 4.246

2.  Genotype-phenotype associations in Sotos syndrome: an analysis of 266 individuals with NSD1 aberrations.

Authors:  Katrina Tatton-Brown; Jenny Douglas; Kim Coleman; Genevieve Baujat; Trevor R P Cole; Soma Das; Denise Horn; Helen E Hughes; I Karen Temple; Francesca Faravelli; Darrel Waggoner; Seval Turkmen; Valerie Cormier-Daire; Alexandre Irrthum; Nazneen Rahman
Journal:  Am J Hum Genet       Date:  2005-06-07       Impact factor: 11.025

3.  Sotos syndrome is associated with deregulation of the MAPK/ERK-signaling pathway.

Authors:  Remco Visser; Ellie B M Landman; Jelle Goeman; Jan M Wit; Marcel Karperien
Journal:  PLoS One       Date:  2012-11-14       Impact factor: 3.240

Review 4.  Sotos syndrome.

Authors:  Geneviève Baujat; Valérie Cormier-Daire
Journal:  Orphanet J Rare Dis       Date:  2007-09-07       Impact factor: 4.123

5.  Expanding the Clinical Spectrum of Sotos Syndrome in a Patient with the New "c.[5867T>A]+[=]"; "p.[Leu1956Gln]+[=]" NSD1 Missense Mutation and Complex Skin Hamartoma.

Authors:  Annalisa Mencarelli; Paolo Prontera; Amedea Mencarelli; Daniela Rogaia; Gabriela Stangoni; Massimiliano Cecconi; Susanna Esposito
Journal:  Int J Mol Sci       Date:  2018-10-16       Impact factor: 5.923

  5 in total

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