| Literature DB >> 15356205 |
Chihiro Akazawa1, Hayami Tsuzuki, Yasuko Nakamura, Yo Sasaki, Kanae Ohsaki, Shun Nakamura, Yoshihiro Arakawa, Shinichi Kohsaka.
Abstract
Nerve injury leads to the induction of a large number of genes to repair the damage and to restore synaptic transmission. We have attempted to identify molecules whose mRNA expression is altered in response to facial nerve axotomy. Here we report that facial nerve axotomy upregulates Sonic hedgehog (Shh) and its receptor Smoothened (Smo) in facial motor neurons of adult rats, whereas facial nerve axotomy does not upregulate mRNA of Shh or Smo in neonatal rats. We tested whether overexpression of Shh in facial motor neurons of axotomized neonatal rats may promote neuronal survival. Adenovirus-mediated overexpression of Shh, but not that of beta-galactosidase, transiently rescues axotomy-induced neuronal cell death for 3-5 d after axotomy. Finally, the pharmacological inhibitor of Shh signaling, cyclopamine, induces motor neuron death in adult rats after axotomy. These results suggest that Shh plays a regulatory role in nerve injury.Entities:
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Year: 2004 PMID: 15356205 PMCID: PMC6729937 DOI: 10.1523/JNEUROSCI.1784-04.2004
Source DB: PubMed Journal: J Neurosci ISSN: 0270-6474 Impact factor: 6.167